Phospholipid Metabolism in Apoptosis:
The last few years have witnessed an explosion of both interest and knowledge about apoptosis, the process by which a cell actively commits suicide. The number of publications on the topic has increased from nothing in the early 1980s to more than 10,000 papers annually today. It is now well recogni...
Gespeichert in:
| Weitere Verfasser: | , |
|---|---|
| Format: | Elektronisch E-Book |
| Sprache: | English |
| Veröffentlicht: |
Boston, MA
Springer US
2002
|
| Schriftenreihe: | Subcellular Biochemistry
36 |
| Schlagworte: | |
| Online-Zugang: | UBR01 URL des Erstveröffentlichers |
| Zusammenfassung: | The last few years have witnessed an explosion of both interest and knowledge about apoptosis, the process by which a cell actively commits suicide. The number of publications on the topic has increased from nothing in the early 1980s to more than 10,000 papers annually today. It is now well recognized that apoptosis is essential in many aspects of normal development and is required for maintaining tissue homeostasis. The idea that life requires death seems somewhat paradoxical, but cell suicide is essential for an animal to survive. For example, without selective destruction of "non-self" T cells, an animal would lack immunity. Similarly, meaningful neural connections in the brain are whittled from a mass of cells. Further, developmental cell remodeling during tissue maturation involves programmed cell death as the major mechanism for functional and structural safe transition of undifferentiated cells to more specialized counterparts. Apoptosis research, with roots in biochemistry, developmental and cell biology, genetics, and immunology, embraces this long-ignored natural law. Failure to properly regulate apoptosis can have catastrophic consequences. Cancer and many diseases (AIDS, Alzheimer's disease, Parkinson's disease, heart attack, stroke, etc. ) are thought to arise from deregulation of apoptosis. As apoptosis emerges as a key biological regulatory mechanism, it has become harder and harder to keep up with new developments in this field |
| Beschreibung: | 1 Online-Ressource (XVI, 340 p. 30 illus) |
| ISBN: | 9780306479311 |
| DOI: | 10.1007/b105359 |
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| 520 | |a The last few years have witnessed an explosion of both interest and knowledge about apoptosis, the process by which a cell actively commits suicide. The number of publications on the topic has increased from nothing in the early 1980s to more than 10,000 papers annually today. It is now well recognized that apoptosis is essential in many aspects of normal development and is required for maintaining tissue homeostasis. The idea that life requires death seems somewhat paradoxical, but cell suicide is essential for an animal to survive. For example, without selective destruction of "non-self" T cells, an animal would lack immunity. Similarly, meaningful neural connections in the brain are whittled from a mass of cells. Further, developmental cell remodeling during tissue maturation involves programmed cell death as the major mechanism for functional and structural safe transition of undifferentiated cells to more specialized counterparts. Apoptosis research, with roots in biochemistry, developmental and cell biology, genetics, and immunology, embraces this long-ignored natural law. Failure to properly regulate apoptosis can have catastrophic consequences. Cancer and many diseases (AIDS, Alzheimer's disease, Parkinson's disease, heart attack, stroke, etc. ) are thought to arise from deregulation of apoptosis. As apoptosis emerges as a key biological regulatory mechanism, it has become harder and harder to keep up with new developments in this field | ||
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| spelling | Phospholipid Metabolism in Apoptosis edited by Peter J. Quinn, Valerian E. Kagan Boston, MA Springer US 2002 1 Online-Ressource (XVI, 340 p. 30 illus) txt rdacontent c rdamedia cr rdacarrier Subcellular Biochemistry 36 The last few years have witnessed an explosion of both interest and knowledge about apoptosis, the process by which a cell actively commits suicide. The number of publications on the topic has increased from nothing in the early 1980s to more than 10,000 papers annually today. It is now well recognized that apoptosis is essential in many aspects of normal development and is required for maintaining tissue homeostasis. The idea that life requires death seems somewhat paradoxical, but cell suicide is essential for an animal to survive. For example, without selective destruction of "non-self" T cells, an animal would lack immunity. Similarly, meaningful neural connections in the brain are whittled from a mass of cells. Further, developmental cell remodeling during tissue maturation involves programmed cell death as the major mechanism for functional and structural safe transition of undifferentiated cells to more specialized counterparts. Apoptosis research, with roots in biochemistry, developmental and cell biology, genetics, and immunology, embraces this long-ignored natural law. Failure to properly regulate apoptosis can have catastrophic consequences. Cancer and many diseases (AIDS, Alzheimer's disease, Parkinson's disease, heart attack, stroke, etc. ) are thought to arise from deregulation of apoptosis. As apoptosis emerges as a key biological regulatory mechanism, it has become harder and harder to keep up with new developments in this field Biochemistry, general Anatomy Human Genetics Biochemistry Human anatomy Human genetics Apoptosis (DE-588)4291096-1 gnd rswk-swf Lipidmembran (DE-588)4167790-0 gnd rswk-swf Phospholipide (DE-588)4131432-3 gnd rswk-swf (DE-588)4143413-4 Aufsatzsammlung gnd-content Apoptosis (DE-588)4291096-1 s Phospholipide (DE-588)4131432-3 s DE-604 Lipidmembran (DE-588)4167790-0 s Quinn, Peter J. edt Kagan, Valerian E. edt Erscheint auch als Druck-Ausgabe 9781475782165 Erscheint auch als Druck-Ausgabe 9780306467820 Erscheint auch als Druck-Ausgabe 9781475782158 https://doi.org/10.1007/b105359 Verlag URL des Erstveröffentlichers Volltext |
| spellingShingle | Phospholipid Metabolism in Apoptosis Biochemistry, general Anatomy Human Genetics Biochemistry Human anatomy Human genetics Apoptosis (DE-588)4291096-1 gnd Lipidmembran (DE-588)4167790-0 gnd Phospholipide (DE-588)4131432-3 gnd |
| subject_GND | (DE-588)4291096-1 (DE-588)4167790-0 (DE-588)4131432-3 (DE-588)4143413-4 |
| title | Phospholipid Metabolism in Apoptosis |
| title_auth | Phospholipid Metabolism in Apoptosis |
| title_exact_search | Phospholipid Metabolism in Apoptosis |
| title_full | Phospholipid Metabolism in Apoptosis edited by Peter J. Quinn, Valerian E. Kagan |
| title_fullStr | Phospholipid Metabolism in Apoptosis edited by Peter J. Quinn, Valerian E. Kagan |
| title_full_unstemmed | Phospholipid Metabolism in Apoptosis edited by Peter J. Quinn, Valerian E. Kagan |
| title_short | Phospholipid Metabolism in Apoptosis |
| title_sort | phospholipid metabolism in apoptosis |
| topic | Biochemistry, general Anatomy Human Genetics Biochemistry Human anatomy Human genetics Apoptosis (DE-588)4291096-1 gnd Lipidmembran (DE-588)4167790-0 gnd Phospholipide (DE-588)4131432-3 gnd |
| topic_facet | Biochemistry, general Anatomy Human Genetics Biochemistry Human anatomy Human genetics Apoptosis Lipidmembran Phospholipide Aufsatzsammlung |
| url | https://doi.org/10.1007/b105359 |
| work_keys_str_mv | AT quinnpeterj phospholipidmetabolisminapoptosis AT kaganvaleriane phospholipidmetabolisminapoptosis |