Multiple Organ Failure: Pathophysiology, Prevention, and Therapy
Inflammation in itself is not to be considered as a disease . . . and in disease, where it can alter the diseased mode of action, it likewise leads to a cure; but where it cannot accomplish that solitary purpose . . . it does mischief - John Hunter, A Treatise on the Blood, ITfIlammation, and Gunsho...
Gespeichert in:
| Weitere Verfasser: | , , |
|---|---|
| Format: | Elektronisch E-Book |
| Sprache: | English |
| Veröffentlicht: |
New York, NY
Springer New York
2000
|
| Schlagworte: | |
| Online-Zugang: | UBR01 Volltext |
| Zusammenfassung: | Inflammation in itself is not to be considered as a disease . . . and in disease, where it can alter the diseased mode of action, it likewise leads to a cure; but where it cannot accomplish that solitary purpose . . . it does mischief - John Hunter, A Treatise on the Blood, ITfIlammation, and Gunshot Woundr (London, 1794)1 As we reached the millennium, we recognized the gap between our scientific knowledge of biologic processes and our more limited clinical capabilities in the care of patients. Our science is strong. Molecular biology is powerful, but our therapy to help patients is weaker and more limited. For this reason, this book focuses on the problems of multiple organ failure (MOF), multiple organ dysfunction syndrome (MODS), and systemic inflammatory response syndrome is, patients who have severe injuries; require major, (SIRS) in high-risk patients, that overwhelming operations; or have serious illnesses requiring intensive care; patients who have diseases elsewhere, in other organs or systems, that limit their capabilities to survive a new insult; and patients who are elderly or at high risk for sepsis or other complications. These are the patients who need our help. They need the advances in science, in molecular biology, immunology, pathophysiology, biochemistry, genetics, high technology, and other areas of maximum support at the bedside. These advances could potentially have the greatest impact on improving patient care |
| Beschreibung: | 1 Online-Ressource (XXII, 712 p) |
| ISBN: | 9781461212225 |
| DOI: | 10.1007/978-1-4612-1222-5 |
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| 520 | |a Inflammation in itself is not to be considered as a disease . . . and in disease, where it can alter the diseased mode of action, it likewise leads to a cure; but where it cannot accomplish that solitary purpose . . . it does mischief - John Hunter, A Treatise on the Blood, ITfIlammation, and Gunshot Woundr (London, 1794)1 As we reached the millennium, we recognized the gap between our scientific knowledge of biologic processes and our more limited clinical capabilities in the care of patients. Our science is strong. Molecular biology is powerful, but our therapy to help patients is weaker and more limited. For this reason, this book focuses on the problems of multiple organ failure (MOF), multiple organ dysfunction syndrome (MODS), and systemic inflammatory response syndrome is, patients who have severe injuries; require major, (SIRS) in high-risk patients, that overwhelming operations; or have serious illnesses requiring intensive care; patients who have diseases elsewhere, in other organs or systems, that limit their capabilities to survive a new insult; and patients who are elderly or at high risk for sepsis or other complications. These are the patients who need our help. They need the advances in science, in molecular biology, immunology, pathophysiology, biochemistry, genetics, high technology, and other areas of maximum support at the bedside. These advances could potentially have the greatest impact on improving patient care | ||
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| spelling | Multiple Organ Failure Pathophysiology, Prevention, and Therapy edited by Arthur E. Baue, Eugen Faist, Donald E. Fry New York, NY Springer New York 2000 1 Online-Ressource (XXII, 712 p) txt rdacontent c rdamedia cr rdacarrier Inflammation in itself is not to be considered as a disease . . . and in disease, where it can alter the diseased mode of action, it likewise leads to a cure; but where it cannot accomplish that solitary purpose . . . it does mischief - John Hunter, A Treatise on the Blood, ITfIlammation, and Gunshot Woundr (London, 1794)1 As we reached the millennium, we recognized the gap between our scientific knowledge of biologic processes and our more limited clinical capabilities in the care of patients. Our science is strong. Molecular biology is powerful, but our therapy to help patients is weaker and more limited. For this reason, this book focuses on the problems of multiple organ failure (MOF), multiple organ dysfunction syndrome (MODS), and systemic inflammatory response syndrome is, patients who have severe injuries; require major, (SIRS) in high-risk patients, that overwhelming operations; or have serious illnesses requiring intensive care; patients who have diseases elsewhere, in other organs or systems, that limit their capabilities to survive a new insult; and patients who are elderly or at high risk for sepsis or other complications. These are the patients who need our help. They need the advances in science, in molecular biology, immunology, pathophysiology, biochemistry, genetics, high technology, and other areas of maximum support at the bedside. These advances could potentially have the greatest impact on improving patient care Surgery Pathology Internal Medicine Intensive / Critical Care Medicine Internal medicine Critical care medicine Multiorganversagen (DE-588)4334293-0 gnd rswk-swf (DE-588)4143413-4 Aufsatzsammlung gnd-content Multiorganversagen (DE-588)4334293-0 s DE-604 Baue, Arthur E. edt Faist, Eugen edt Fry, Donald E. edt Erscheint auch als Druck-Ausgabe 9780387987330 Erscheint auch als Druck-Ausgabe 9781461270492 Erscheint auch als Druck-Ausgabe 9781461212232 https://doi.org/10.1007/978-1-4612-1222-5 Verlag URL des Erstveröffentlichers Volltext |
| spellingShingle | Multiple Organ Failure Pathophysiology, Prevention, and Therapy Surgery Pathology Internal Medicine Intensive / Critical Care Medicine Internal medicine Critical care medicine Multiorganversagen (DE-588)4334293-0 gnd |
| subject_GND | (DE-588)4334293-0 (DE-588)4143413-4 |
| title | Multiple Organ Failure Pathophysiology, Prevention, and Therapy |
| title_auth | Multiple Organ Failure Pathophysiology, Prevention, and Therapy |
| title_exact_search | Multiple Organ Failure Pathophysiology, Prevention, and Therapy |
| title_full | Multiple Organ Failure Pathophysiology, Prevention, and Therapy edited by Arthur E. Baue, Eugen Faist, Donald E. Fry |
| title_fullStr | Multiple Organ Failure Pathophysiology, Prevention, and Therapy edited by Arthur E. Baue, Eugen Faist, Donald E. Fry |
| title_full_unstemmed | Multiple Organ Failure Pathophysiology, Prevention, and Therapy edited by Arthur E. Baue, Eugen Faist, Donald E. Fry |
| title_short | Multiple Organ Failure |
| title_sort | multiple organ failure pathophysiology prevention and therapy |
| title_sub | Pathophysiology, Prevention, and Therapy |
| topic | Surgery Pathology Internal Medicine Intensive / Critical Care Medicine Internal medicine Critical care medicine Multiorganversagen (DE-588)4334293-0 gnd |
| topic_facet | Surgery Pathology Internal Medicine Intensive / Critical Care Medicine Internal medicine Critical care medicine Multiorganversagen Aufsatzsammlung |
| url | https://doi.org/10.1007/978-1-4612-1222-5 |
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