Cardiovascular Specific Gene Expression:
Improving our insights into the genetic predisposition to cardiovascular disease is one of the most important challenges in our field in the next millennium, not only to unravel the cause of disease but also to improve the selection of patients for particular treatments. Nowadays, for example, subje...
Gespeichert in:
| Weitere Verfasser: | , , |
|---|---|
| Format: | Elektronisch E-Book |
| Sprache: | English |
| Veröffentlicht: |
Dordrecht
Springer Netherlands
1999
|
| Schriftenreihe: | Developments in Cardiovascular Medicine
214 |
| Schlagworte: | |
| Online-Zugang: | UBR01 Volltext |
| Zusammenfassung: | Improving our insights into the genetic predisposition to cardiovascular disease is one of the most important challenges in our field in the next millennium, not only to unravel the cause of disease but also to improve the selection of patients for particular treatments. Nowadays, for example, subjects with a cholesterol above a particular plasma level are exposed to a cholesterol lowering regime based upon the beneficial outcome of epidemiological studies which include subjects not prone to the disease, despite a plasma cholesterol above the accepted level. Identification of the patients who are genetically predisposed to the consequences of this disorder will reduce the number of subjects unnecessarily treated and, hence, the costs of health care. Because in most cardiovascular diseases the genetic component is a consequence of more than one gene defect, only limited progress has as yet been made in identifying subjects genetically at risk. For example, in hypertension only in less than 10% of the patients the genetic defect has been identified. It has been known for quite some time that in heart and blood vessels fetal genes are as high blood pressure and upregulated or induced when they are exposed to such disorders ischemia. Little is known about the function of these genes in the cardiac and vascular adaptation to these disorders; only guesses can be made |
| Beschreibung: | 1 Online-Ressource (XIV, 330 p) |
| ISBN: | 9789401593212 |
| DOI: | 10.1007/978-94-015-9321-2 |
Internformat
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| 520 | |a Improving our insights into the genetic predisposition to cardiovascular disease is one of the most important challenges in our field in the next millennium, not only to unravel the cause of disease but also to improve the selection of patients for particular treatments. Nowadays, for example, subjects with a cholesterol above a particular plasma level are exposed to a cholesterol lowering regime based upon the beneficial outcome of epidemiological studies which include subjects not prone to the disease, despite a plasma cholesterol above the accepted level. Identification of the patients who are genetically predisposed to the consequences of this disorder will reduce the number of subjects unnecessarily treated and, hence, the costs of health care. Because in most cardiovascular diseases the genetic component is a consequence of more than one gene defect, only limited progress has as yet been made in identifying subjects genetically at risk. For example, in hypertension only in less than 10% of the patients the genetic defect has been identified. It has been known for quite some time that in heart and blood vessels fetal genes are as high blood pressure and upregulated or induced when they are exposed to such disorders ischemia. Little is known about the function of these genes in the cardiac and vascular adaptation to these disorders; only guesses can be made | ||
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Datensatz im Suchindex
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| discipline | Medizin |
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| spelling | Cardiovascular Specific Gene Expression edited by Pieter A. Doevendans, Robert S. Reneman, Marc van Bilsen Dordrecht Springer Netherlands 1999 1 Online-Ressource (XIV, 330 p) txt rdacontent c rdamedia cr rdacarrier Developments in Cardiovascular Medicine 214 Improving our insights into the genetic predisposition to cardiovascular disease is one of the most important challenges in our field in the next millennium, not only to unravel the cause of disease but also to improve the selection of patients for particular treatments. Nowadays, for example, subjects with a cholesterol above a particular plasma level are exposed to a cholesterol lowering regime based upon the beneficial outcome of epidemiological studies which include subjects not prone to the disease, despite a plasma cholesterol above the accepted level. Identification of the patients who are genetically predisposed to the consequences of this disorder will reduce the number of subjects unnecessarily treated and, hence, the costs of health care. Because in most cardiovascular diseases the genetic component is a consequence of more than one gene defect, only limited progress has as yet been made in identifying subjects genetically at risk. For example, in hypertension only in less than 10% of the patients the genetic defect has been identified. It has been known for quite some time that in heart and blood vessels fetal genes are as high blood pressure and upregulated or induced when they are exposed to such disorders ischemia. Little is known about the function of these genes in the cardiac and vascular adaptation to these disorders; only guesses can be made Cardiology Animal Anatomy / Morphology / Histology Morphology (Animals) Molekularpathologie (DE-588)4170396-0 gnd rswk-swf Genexpression (DE-588)4020136-3 gnd rswk-swf Gentherapie (DE-588)4296957-8 gnd rswk-swf Kardiovaskuläre Krankheit (DE-588)4024666-8 gnd rswk-swf 1\p (DE-588)1071861417 Konferenzschrift gnd-content Kardiovaskuläre Krankheit (DE-588)4024666-8 s Genexpression (DE-588)4020136-3 s 2\p DE-604 Molekularpathologie (DE-588)4170396-0 s 3\p DE-604 Gentherapie (DE-588)4296957-8 s 4\p DE-604 Doevendans, Pieter A. edt Reneman, Robert S. edt Bilsen, Marc van edt Erscheint auch als Druck-Ausgabe 9789048151899 Erscheint auch als Druck-Ausgabe 9780792356332 Erscheint auch als Druck-Ausgabe 9789401593229 https://doi.org/10.1007/978-94-015-9321-2 Verlag URL des Erstveröffentlichers Volltext 1\p cgwrk 20201028 DE-101 https://d-nb.info/provenance/plan#cgwrk 2\p cgwrk 20201028 DE-101 https://d-nb.info/provenance/plan#cgwrk 3\p cgwrk 20201028 DE-101 https://d-nb.info/provenance/plan#cgwrk 4\p cgwrk 20201028 DE-101 https://d-nb.info/provenance/plan#cgwrk |
| spellingShingle | Cardiovascular Specific Gene Expression Cardiology Animal Anatomy / Morphology / Histology Morphology (Animals) Molekularpathologie (DE-588)4170396-0 gnd Genexpression (DE-588)4020136-3 gnd Gentherapie (DE-588)4296957-8 gnd Kardiovaskuläre Krankheit (DE-588)4024666-8 gnd |
| subject_GND | (DE-588)4170396-0 (DE-588)4020136-3 (DE-588)4296957-8 (DE-588)4024666-8 (DE-588)1071861417 |
| title | Cardiovascular Specific Gene Expression |
| title_auth | Cardiovascular Specific Gene Expression |
| title_exact_search | Cardiovascular Specific Gene Expression |
| title_full | Cardiovascular Specific Gene Expression edited by Pieter A. Doevendans, Robert S. Reneman, Marc van Bilsen |
| title_fullStr | Cardiovascular Specific Gene Expression edited by Pieter A. Doevendans, Robert S. Reneman, Marc van Bilsen |
| title_full_unstemmed | Cardiovascular Specific Gene Expression edited by Pieter A. Doevendans, Robert S. Reneman, Marc van Bilsen |
| title_short | Cardiovascular Specific Gene Expression |
| title_sort | cardiovascular specific gene expression |
| topic | Cardiology Animal Anatomy / Morphology / Histology Morphology (Animals) Molekularpathologie (DE-588)4170396-0 gnd Genexpression (DE-588)4020136-3 gnd Gentherapie (DE-588)4296957-8 gnd Kardiovaskuläre Krankheit (DE-588)4024666-8 gnd |
| topic_facet | Cardiology Animal Anatomy / Morphology / Histology Morphology (Animals) Molekularpathologie Genexpression Gentherapie Kardiovaskuläre Krankheit Konferenzschrift |
| url | https://doi.org/10.1007/978-94-015-9321-2 |
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