Cardiac Remodeling and Failure:
According to the World Health Report (2000 http:/ /www. who. int/whr), of the 55 million deaths worldwide in 1999, more than 16 million were secondary to car diovascular complications. With the prospect of world population increasing from the current level of 6 billion to 9 billion by the middle of...
Gespeichert in:
Weitere Verfasser: | , , , |
---|---|
Format: | Elektronisch E-Book |
Sprache: | English |
Veröffentlicht: |
Boston, MA
Springer US
2003
|
Schriftenreihe: | Progress in Experimental Cardiology
5 |
Schlagworte: | |
Online-Zugang: | UBR01 Volltext |
Zusammenfassung: | According to the World Health Report (2000 http:/ /www. who. int/whr), of the 55 million deaths worldwide in 1999, more than 16 million were secondary to car diovascular complications. With the prospect of world population increasing from the current level of 6 billion to 9 billion by the middle of this century, the burden of cardiac disease is going to increase astronomically. Furthermore, scientists are being challenged not only to reduce mortality, but also to improve quality of life. Thus, more than ever, intellectuals from different disciplines including biology, sociology, informatics and health care have to join forces to meet the mandate. The World Heart Congress with a focus on "Frontiers in Cardiovascular Health" held in Winnipeg during July 6-11, 2001, made a unique attempt to bring these specialists together to brainstorm and map out the course of action for cardiovascular research and health in the next century. Anytime there is a relative increase in the workload on the heart, there are adap tive myocardial as well as humoral responses. When these adaptations or remodel ing at the organ, subcellular or gene level, become inadequate for a proper tissue perfusion, the condition of heart failure ensues. Prevention of the factors leading to the relative increase in workload as well as a better understanding of the adap tive responses and their failure are some of the hopes to combat the morbidity and mortality due to heart failure |
Beschreibung: | 1 Online-Ressource (XVI, 545 p) |
ISBN: | 9781441992628 |
DOI: | 10.1007/978-1-4419-9262-8 |
Internformat
MARC
LEADER | 00000nmm a2200000zcb4500 | ||
---|---|---|---|
001 | BV046145376 | ||
003 | DE-604 | ||
005 | 00000000000000.0 | ||
007 | cr|uuu---uuuuu | ||
008 | 190905s2003 |||| o||u| ||||||eng d | ||
020 | |a 9781441992628 |9 978-1-4419-9262-8 | ||
024 | 7 | |a 10.1007/978-1-4419-9262-8 |2 doi | |
035 | |a (ZDB-2-SME)978-1-4419-9262-8 | ||
035 | |a (OCoLC)1119101581 | ||
035 | |a (DE-599)BVBBV046145376 | ||
040 | |a DE-604 |b ger |e aacr | ||
041 | 0 | |a eng | |
049 | |a DE-355 | ||
082 | 0 | |a 616.12 |2 23 | |
245 | 1 | 0 | |a Cardiac Remodeling and Failure |c edited by Pawan K. Singal, Ian M. C. Dixon, Lorrie A. Kirshenbaum, Naranjan S. Dhalla |
264 | 1 | |a Boston, MA |b Springer US |c 2003 | |
300 | |a 1 Online-Ressource (XVI, 545 p) | ||
336 | |b txt |2 rdacontent | ||
337 | |b c |2 rdamedia | ||
338 | |b cr |2 rdacarrier | ||
490 | 0 | |a Progress in Experimental Cardiology |v 5 | |
520 | |a According to the World Health Report (2000 http:/ /www. who. int/whr), of the 55 million deaths worldwide in 1999, more than 16 million were secondary to car diovascular complications. With the prospect of world population increasing from the current level of 6 billion to 9 billion by the middle of this century, the burden of cardiac disease is going to increase astronomically. Furthermore, scientists are being challenged not only to reduce mortality, but also to improve quality of life. Thus, more than ever, intellectuals from different disciplines including biology, sociology, informatics and health care have to join forces to meet the mandate. The World Heart Congress with a focus on "Frontiers in Cardiovascular Health" held in Winnipeg during July 6-11, 2001, made a unique attempt to bring these specialists together to brainstorm and map out the course of action for cardiovascular research and health in the next century. Anytime there is a relative increase in the workload on the heart, there are adap tive myocardial as well as humoral responses. When these adaptations or remodel ing at the organ, subcellular or gene level, become inadequate for a proper tissue perfusion, the condition of heart failure ensues. Prevention of the factors leading to the relative increase in workload as well as a better understanding of the adap tive responses and their failure are some of the hopes to combat the morbidity and mortality due to heart failure | ||
650 | 4 | |a Cardiology | |
650 | 4 | |a Cardiology | |
700 | 1 | |a Singal, Pawan K. |4 edt | |
700 | 1 | |a Dixon, Ian M. C. |4 edt | |
700 | 1 | |a Kirshenbaum, Lorrie A. |4 edt | |
700 | 1 | |a Dhalla, Naranjan S. |4 edt | |
776 | 0 | 8 | |i Erscheint auch als |n Druck-Ausgabe |z 9781402071775 |
776 | 0 | 8 | |i Erscheint auch als |n Druck-Ausgabe |z 9781441992635 |
776 | 0 | 8 | |i Erscheint auch als |n Druck-Ausgabe |z 9781461348641 |
856 | 4 | 0 | |u https://doi.org/10.1007/978-1-4419-9262-8 |x Verlag |z URL des Erstveröffentlichers |3 Volltext |
912 | |a ZDB-2-SME | ||
940 | 1 | |q ZDB-2-SME_1990/2004 | |
999 | |a oai:aleph.bib-bvb.de:BVB01-031525561 | ||
966 | e | |u https://doi.org/10.1007/978-1-4419-9262-8 |l UBR01 |p ZDB-2-SME |q ZDB-2-SME_1990/2004 |x Verlag |3 Volltext |
Datensatz im Suchindex
_version_ | 1804180481964507136 |
---|---|
any_adam_object | |
author2 | Singal, Pawan K. Dixon, Ian M. C. Kirshenbaum, Lorrie A. Dhalla, Naranjan S. |
author2_role | edt edt edt edt |
author2_variant | p k s pk pks i m c d imc imcd l a k la lak n s d ns nsd |
author_facet | Singal, Pawan K. Dixon, Ian M. C. Kirshenbaum, Lorrie A. Dhalla, Naranjan S. |
building | Verbundindex |
bvnumber | BV046145376 |
collection | ZDB-2-SME |
ctrlnum | (ZDB-2-SME)978-1-4419-9262-8 (OCoLC)1119101581 (DE-599)BVBBV046145376 |
dewey-full | 616.12 |
dewey-hundreds | 600 - Technology (Applied sciences) |
dewey-ones | 616 - Diseases |
dewey-raw | 616.12 |
dewey-search | 616.12 |
dewey-sort | 3616.12 |
dewey-tens | 610 - Medicine and health |
discipline | Medizin |
doi_str_mv | 10.1007/978-1-4419-9262-8 |
format | Electronic eBook |
fullrecord | <?xml version="1.0" encoding="UTF-8"?><collection xmlns="http://www.loc.gov/MARC21/slim"><record><leader>03106nmm a2200457zcb4500</leader><controlfield tag="001">BV046145376</controlfield><controlfield tag="003">DE-604</controlfield><controlfield tag="005">00000000000000.0</controlfield><controlfield tag="007">cr|uuu---uuuuu</controlfield><controlfield tag="008">190905s2003 |||| o||u| ||||||eng d</controlfield><datafield tag="020" ind1=" " ind2=" "><subfield code="a">9781441992628</subfield><subfield code="9">978-1-4419-9262-8</subfield></datafield><datafield tag="024" ind1="7" ind2=" "><subfield code="a">10.1007/978-1-4419-9262-8</subfield><subfield code="2">doi</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(ZDB-2-SME)978-1-4419-9262-8</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(OCoLC)1119101581</subfield></datafield><datafield tag="035" ind1=" " ind2=" "><subfield code="a">(DE-599)BVBBV046145376</subfield></datafield><datafield tag="040" ind1=" " ind2=" "><subfield code="a">DE-604</subfield><subfield code="b">ger</subfield><subfield code="e">aacr</subfield></datafield><datafield tag="041" ind1="0" ind2=" "><subfield code="a">eng</subfield></datafield><datafield tag="049" ind1=" " ind2=" "><subfield code="a">DE-355</subfield></datafield><datafield tag="082" ind1="0" ind2=" "><subfield code="a">616.12</subfield><subfield code="2">23</subfield></datafield><datafield tag="245" ind1="1" ind2="0"><subfield code="a">Cardiac Remodeling and Failure</subfield><subfield code="c">edited by Pawan K. Singal, Ian M. C. Dixon, Lorrie A. Kirshenbaum, Naranjan S. Dhalla</subfield></datafield><datafield tag="264" ind1=" " ind2="1"><subfield code="a">Boston, MA</subfield><subfield code="b">Springer US</subfield><subfield code="c">2003</subfield></datafield><datafield tag="300" ind1=" " ind2=" "><subfield code="a">1 Online-Ressource (XVI, 545 p)</subfield></datafield><datafield tag="336" ind1=" " ind2=" "><subfield code="b">txt</subfield><subfield code="2">rdacontent</subfield></datafield><datafield tag="337" ind1=" " ind2=" "><subfield code="b">c</subfield><subfield code="2">rdamedia</subfield></datafield><datafield tag="338" ind1=" " ind2=" "><subfield code="b">cr</subfield><subfield code="2">rdacarrier</subfield></datafield><datafield tag="490" ind1="0" ind2=" "><subfield code="a">Progress in Experimental Cardiology</subfield><subfield code="v">5</subfield></datafield><datafield tag="520" ind1=" " ind2=" "><subfield code="a">According to the World Health Report (2000 http:/ /www. who. int/whr), of the 55 million deaths worldwide in 1999, more than 16 million were secondary to car diovascular complications. With the prospect of world population increasing from the current level of 6 billion to 9 billion by the middle of this century, the burden of cardiac disease is going to increase astronomically. Furthermore, scientists are being challenged not only to reduce mortality, but also to improve quality of life. Thus, more than ever, intellectuals from different disciplines including biology, sociology, informatics and health care have to join forces to meet the mandate. The World Heart Congress with a focus on "Frontiers in Cardiovascular Health" held in Winnipeg during July 6-11, 2001, made a unique attempt to bring these specialists together to brainstorm and map out the course of action for cardiovascular research and health in the next century. Anytime there is a relative increase in the workload on the heart, there are adap tive myocardial as well as humoral responses. When these adaptations or remodel ing at the organ, subcellular or gene level, become inadequate for a proper tissue perfusion, the condition of heart failure ensues. Prevention of the factors leading to the relative increase in workload as well as a better understanding of the adap tive responses and their failure are some of the hopes to combat the morbidity and mortality due to heart failure</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Cardiology</subfield></datafield><datafield tag="650" ind1=" " ind2="4"><subfield code="a">Cardiology</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Singal, Pawan K.</subfield><subfield code="4">edt</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Dixon, Ian M. C.</subfield><subfield code="4">edt</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Kirshenbaum, Lorrie A.</subfield><subfield code="4">edt</subfield></datafield><datafield tag="700" ind1="1" ind2=" "><subfield code="a">Dhalla, Naranjan S.</subfield><subfield code="4">edt</subfield></datafield><datafield tag="776" ind1="0" ind2="8"><subfield code="i">Erscheint auch als</subfield><subfield code="n">Druck-Ausgabe</subfield><subfield code="z">9781402071775</subfield></datafield><datafield tag="776" ind1="0" ind2="8"><subfield code="i">Erscheint auch als</subfield><subfield code="n">Druck-Ausgabe</subfield><subfield code="z">9781441992635</subfield></datafield><datafield tag="776" ind1="0" ind2="8"><subfield code="i">Erscheint auch als</subfield><subfield code="n">Druck-Ausgabe</subfield><subfield code="z">9781461348641</subfield></datafield><datafield tag="856" ind1="4" ind2="0"><subfield code="u">https://doi.org/10.1007/978-1-4419-9262-8</subfield><subfield code="x">Verlag</subfield><subfield code="z">URL des Erstveröffentlichers</subfield><subfield code="3">Volltext</subfield></datafield><datafield tag="912" ind1=" " ind2=" "><subfield code="a">ZDB-2-SME</subfield></datafield><datafield tag="940" ind1="1" ind2=" "><subfield code="q">ZDB-2-SME_1990/2004</subfield></datafield><datafield tag="999" ind1=" " ind2=" "><subfield code="a">oai:aleph.bib-bvb.de:BVB01-031525561</subfield></datafield><datafield tag="966" ind1="e" ind2=" "><subfield code="u">https://doi.org/10.1007/978-1-4419-9262-8</subfield><subfield code="l">UBR01</subfield><subfield code="p">ZDB-2-SME</subfield><subfield code="q">ZDB-2-SME_1990/2004</subfield><subfield code="x">Verlag</subfield><subfield code="3">Volltext</subfield></datafield></record></collection> |
id | DE-604.BV046145376 |
illustrated | Not Illustrated |
indexdate | 2024-07-10T08:36:27Z |
institution | BVB |
isbn | 9781441992628 |
language | English |
oai_aleph_id | oai:aleph.bib-bvb.de:BVB01-031525561 |
oclc_num | 1119101581 |
open_access_boolean | |
owner | DE-355 DE-BY-UBR |
owner_facet | DE-355 DE-BY-UBR |
physical | 1 Online-Ressource (XVI, 545 p) |
psigel | ZDB-2-SME ZDB-2-SME_1990/2004 ZDB-2-SME ZDB-2-SME_1990/2004 |
publishDate | 2003 |
publishDateSearch | 2003 |
publishDateSort | 2003 |
publisher | Springer US |
record_format | marc |
series2 | Progress in Experimental Cardiology |
spelling | Cardiac Remodeling and Failure edited by Pawan K. Singal, Ian M. C. Dixon, Lorrie A. Kirshenbaum, Naranjan S. Dhalla Boston, MA Springer US 2003 1 Online-Ressource (XVI, 545 p) txt rdacontent c rdamedia cr rdacarrier Progress in Experimental Cardiology 5 According to the World Health Report (2000 http:/ /www. who. int/whr), of the 55 million deaths worldwide in 1999, more than 16 million were secondary to car diovascular complications. With the prospect of world population increasing from the current level of 6 billion to 9 billion by the middle of this century, the burden of cardiac disease is going to increase astronomically. Furthermore, scientists are being challenged not only to reduce mortality, but also to improve quality of life. Thus, more than ever, intellectuals from different disciplines including biology, sociology, informatics and health care have to join forces to meet the mandate. The World Heart Congress with a focus on "Frontiers in Cardiovascular Health" held in Winnipeg during July 6-11, 2001, made a unique attempt to bring these specialists together to brainstorm and map out the course of action for cardiovascular research and health in the next century. Anytime there is a relative increase in the workload on the heart, there are adap tive myocardial as well as humoral responses. When these adaptations or remodel ing at the organ, subcellular or gene level, become inadequate for a proper tissue perfusion, the condition of heart failure ensues. Prevention of the factors leading to the relative increase in workload as well as a better understanding of the adap tive responses and their failure are some of the hopes to combat the morbidity and mortality due to heart failure Cardiology Singal, Pawan K. edt Dixon, Ian M. C. edt Kirshenbaum, Lorrie A. edt Dhalla, Naranjan S. edt Erscheint auch als Druck-Ausgabe 9781402071775 Erscheint auch als Druck-Ausgabe 9781441992635 Erscheint auch als Druck-Ausgabe 9781461348641 https://doi.org/10.1007/978-1-4419-9262-8 Verlag URL des Erstveröffentlichers Volltext |
spellingShingle | Cardiac Remodeling and Failure Cardiology |
title | Cardiac Remodeling and Failure |
title_auth | Cardiac Remodeling and Failure |
title_exact_search | Cardiac Remodeling and Failure |
title_full | Cardiac Remodeling and Failure edited by Pawan K. Singal, Ian M. C. Dixon, Lorrie A. Kirshenbaum, Naranjan S. Dhalla |
title_fullStr | Cardiac Remodeling and Failure edited by Pawan K. Singal, Ian M. C. Dixon, Lorrie A. Kirshenbaum, Naranjan S. Dhalla |
title_full_unstemmed | Cardiac Remodeling and Failure edited by Pawan K. Singal, Ian M. C. Dixon, Lorrie A. Kirshenbaum, Naranjan S. Dhalla |
title_short | Cardiac Remodeling and Failure |
title_sort | cardiac remodeling and failure |
topic | Cardiology |
topic_facet | Cardiology |
url | https://doi.org/10.1007/978-1-4419-9262-8 |
work_keys_str_mv | AT singalpawank cardiacremodelingandfailure AT dixonianmc cardiacremodelingandfailure AT kirshenbaumlorriea cardiacremodelingandfailure AT dhallanaranjans cardiacremodelingandfailure |