Genetics of Dyslipidemia:
Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common an...
Gespeichert in:
| 1. Verfasser: | |
|---|---|
| Format: | Elektronisch E-Book |
| Sprache: | English |
| Veröffentlicht: |
Boston, MA
Springer US
2001
|
| Schriftenreihe: | Basic Science for the Cardiologist
7 |
| Schlagworte: | |
| Online-Zugang: | UBR01 Volltext |
| Zusammenfassung: | Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease. Dyslipidemia, are disorders of the metabolism of soluble transporters of lipids in extracellular spaces of the human body (including blood), called lipoproteins. They are major cardiovascular risk factors, causally related with atherosclerosis and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors. The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of observations has refined our current knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system (e.g., brain, immune system, and development). These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations |
| Beschreibung: | 1 Online-Ressource (XI, 315 p) |
| ISBN: | 9781461515173 |
| DOI: | 10.1007/978-1-4615-1517-3 |
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| 520 | |a Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease. Dyslipidemia, are disorders of the metabolism of soluble transporters of lipids in extracellular spaces of the human body (including blood), called lipoproteins. They are major cardiovascular risk factors, causally related with atherosclerosis and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors. The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of observations has refined our current knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system (e.g., brain, immune system, and development). These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations | ||
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| author | Benlian, Pascale |
| author_facet | Benlian, Pascale |
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| dewey-full | 616.12 |
| dewey-hundreds | 600 - Technology (Applied sciences) |
| dewey-ones | 616 - Diseases |
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| dewey-sort | 3616.12 |
| dewey-tens | 610 - Medicine and health |
| discipline | Medizin |
| doi_str_mv | 10.1007/978-1-4615-1517-3 |
| format | Electronic eBook |
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| indexdate | 2024-07-10T08:36:26Z |
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| isbn | 9781461515173 |
| language | English |
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| spelling | Benlian, Pascale Verfasser aut Genetics of Dyslipidemia by Pascale Benlian Boston, MA Springer US 2001 1 Online-Ressource (XI, 315 p) txt rdacontent c rdamedia cr rdacarrier Basic Science for the Cardiologist 7 Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease. Dyslipidemia, are disorders of the metabolism of soluble transporters of lipids in extracellular spaces of the human body (including blood), called lipoproteins. They are major cardiovascular risk factors, causally related with atherosclerosis and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors. The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of observations has refined our current knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system (e.g., brain, immune system, and development). These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations Cardiology Human Genetics Biochemistry, general Human genetics Biochemistry Hyperlipidämie (DE-588)4161072-6 gnd rswk-swf Genetik (DE-588)4071711-2 gnd rswk-swf Hyperlipidämie (DE-588)4161072-6 s Genetik (DE-588)4071711-2 s DE-604 Erscheint auch als Druck-Ausgabe 9780792373629 Erscheint auch als Druck-Ausgabe 9781461355939 Erscheint auch als Druck-Ausgabe 9781461515180 https://doi.org/10.1007/978-1-4615-1517-3 Verlag URL des Erstveröffentlichers Volltext |
| spellingShingle | Benlian, Pascale Genetics of Dyslipidemia Cardiology Human Genetics Biochemistry, general Human genetics Biochemistry Hyperlipidämie (DE-588)4161072-6 gnd Genetik (DE-588)4071711-2 gnd |
| subject_GND | (DE-588)4161072-6 (DE-588)4071711-2 |
| title | Genetics of Dyslipidemia |
| title_auth | Genetics of Dyslipidemia |
| title_exact_search | Genetics of Dyslipidemia |
| title_full | Genetics of Dyslipidemia by Pascale Benlian |
| title_fullStr | Genetics of Dyslipidemia by Pascale Benlian |
| title_full_unstemmed | Genetics of Dyslipidemia by Pascale Benlian |
| title_short | Genetics of Dyslipidemia |
| title_sort | genetics of dyslipidemia |
| topic | Cardiology Human Genetics Biochemistry, general Human genetics Biochemistry Hyperlipidämie (DE-588)4161072-6 gnd Genetik (DE-588)4071711-2 gnd |
| topic_facet | Cardiology Human Genetics Biochemistry, general Human genetics Biochemistry Hyperlipidämie Genetik |
| url | https://doi.org/10.1007/978-1-4615-1517-3 |
| work_keys_str_mv | AT benlianpascale geneticsofdyslipidemia |