Pathophysiology of Severe Ischemic Myocardial Injury:
In industrialized countries, ischemic heart disease is by far the most common organ-specific cause of death. The thrombotic occlusion of a coronary artery which had previously been severely altered by atherosclerosis, is the most frequent cause of ischemic deterioration of myocardial tissue, i. e. m...
Gespeichert in:
| Weitere Verfasser: | |
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| Format: | Elektronisch E-Book |
| Sprache: | English |
| Veröffentlicht: |
Dordrecht
Springer Netherlands
1990
|
| Schriftenreihe: | Developments in Cardiovascular Medicine
104 |
| Schlagworte: | |
| Online-Zugang: | UBR01 Volltext |
| Zusammenfassung: | In industrialized countries, ischemic heart disease is by far the most common organ-specific cause of death. The thrombotic occlusion of a coronary artery which had previously been severely altered by atherosclerosis, is the most frequent cause of ischemic deterioration of myocardial tissue, i. e. myocardial infarction. Death of the human individual occurs when myocardial ischemia causes a critical impairment of cardiac pump function. The failure of a heart with an ischemic area may be due to the amount and location of contractile tissue becoming paralyzed or even necrotic, or to arrhythmias provoked by the ischemic condition, or by a combination of both factors. Considerable progress has been made in the development of antiarrythmic therapy. Effective tools have been developed to reperfuse ischemic myocar dial tissue as soon as the patient reaches hospital. However, therapeutical principles for the ischemic-reperfused myocardium which would specifically interfere with the state of injury of the ischemic tissue at the onset of reperfusion, and avoid the apparent hazards of the reperfusion process itself, have yet to be established. But not only approved therapeutical concepts are lacking, the pathophysiology of myocardial cell injury in pro gressive ischemia and under reperfusion is in itself only partly understood |
| Beschreibung: | 1 Online-Ressource (440 p) |
| ISBN: | 9789400904750 |
| DOI: | 10.1007/978-94-009-0475-0 |
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| 520 | |a In industrialized countries, ischemic heart disease is by far the most common organ-specific cause of death. The thrombotic occlusion of a coronary artery which had previously been severely altered by atherosclerosis, is the most frequent cause of ischemic deterioration of myocardial tissue, i. e. myocardial infarction. Death of the human individual occurs when myocardial ischemia causes a critical impairment of cardiac pump function. The failure of a heart with an ischemic area may be due to the amount and location of contractile tissue becoming paralyzed or even necrotic, or to arrhythmias provoked by the ischemic condition, or by a combination of both factors. Considerable progress has been made in the development of antiarrythmic therapy. Effective tools have been developed to reperfuse ischemic myocar dial tissue as soon as the patient reaches hospital. However, therapeutical principles for the ischemic-reperfused myocardium which would specifically interfere with the state of injury of the ischemic tissue at the onset of reperfusion, and avoid the apparent hazards of the reperfusion process itself, have yet to be established. But not only approved therapeutical concepts are lacking, the pathophysiology of myocardial cell injury in pro gressive ischemia and under reperfusion is in itself only partly understood | ||
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Datensatz im Suchindex
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| spelling | Pathophysiology of Severe Ischemic Myocardial Injury edited by Hans Michael Piper Dordrecht Springer Netherlands 1990 1 Online-Ressource (440 p) txt rdacontent c rdamedia cr rdacarrier Developments in Cardiovascular Medicine 104 In industrialized countries, ischemic heart disease is by far the most common organ-specific cause of death. The thrombotic occlusion of a coronary artery which had previously been severely altered by atherosclerosis, is the most frequent cause of ischemic deterioration of myocardial tissue, i. e. myocardial infarction. Death of the human individual occurs when myocardial ischemia causes a critical impairment of cardiac pump function. The failure of a heart with an ischemic area may be due to the amount and location of contractile tissue becoming paralyzed or even necrotic, or to arrhythmias provoked by the ischemic condition, or by a combination of both factors. Considerable progress has been made in the development of antiarrythmic therapy. Effective tools have been developed to reperfuse ischemic myocar dial tissue as soon as the patient reaches hospital. However, therapeutical principles for the ischemic-reperfused myocardium which would specifically interfere with the state of injury of the ischemic tissue at the onset of reperfusion, and avoid the apparent hazards of the reperfusion process itself, have yet to be established. But not only approved therapeutical concepts are lacking, the pathophysiology of myocardial cell injury in pro gressive ischemia and under reperfusion is in itself only partly understood Cardiology Ätiologie (DE-588)4000646-3 gnd rswk-swf Herzkrankheit (DE-588)4024663-2 gnd rswk-swf Ischämie (DE-588)4162456-7 gnd rswk-swf Pathophysiologie (DE-588)4044898-8 gnd rswk-swf (DE-588)4143413-4 Aufsatzsammlung gnd-content Herzkrankheit (DE-588)4024663-2 s Ischämie (DE-588)4162456-7 s Pathophysiologie (DE-588)4044898-8 s DE-604 Ätiologie (DE-588)4000646-3 s Piper, Hans Michael edt Erscheint auch als Druck-Ausgabe 9789401066990 Erscheint auch als Druck-Ausgabe 9780792304593 Erscheint auch als Druck-Ausgabe 9789400904767 https://doi.org/10.1007/978-94-009-0475-0 Verlag URL des Erstveröffentlichers Volltext |
| spellingShingle | Pathophysiology of Severe Ischemic Myocardial Injury Cardiology Ätiologie (DE-588)4000646-3 gnd Herzkrankheit (DE-588)4024663-2 gnd Ischämie (DE-588)4162456-7 gnd Pathophysiologie (DE-588)4044898-8 gnd |
| subject_GND | (DE-588)4000646-3 (DE-588)4024663-2 (DE-588)4162456-7 (DE-588)4044898-8 (DE-588)4143413-4 |
| title | Pathophysiology of Severe Ischemic Myocardial Injury |
| title_auth | Pathophysiology of Severe Ischemic Myocardial Injury |
| title_exact_search | Pathophysiology of Severe Ischemic Myocardial Injury |
| title_full | Pathophysiology of Severe Ischemic Myocardial Injury edited by Hans Michael Piper |
| title_fullStr | Pathophysiology of Severe Ischemic Myocardial Injury edited by Hans Michael Piper |
| title_full_unstemmed | Pathophysiology of Severe Ischemic Myocardial Injury edited by Hans Michael Piper |
| title_short | Pathophysiology of Severe Ischemic Myocardial Injury |
| title_sort | pathophysiology of severe ischemic myocardial injury |
| topic | Cardiology Ätiologie (DE-588)4000646-3 gnd Herzkrankheit (DE-588)4024663-2 gnd Ischämie (DE-588)4162456-7 gnd Pathophysiologie (DE-588)4044898-8 gnd |
| topic_facet | Cardiology Ätiologie Herzkrankheit Ischämie Pathophysiologie Aufsatzsammlung |
| url | https://doi.org/10.1007/978-94-009-0475-0 |
| work_keys_str_mv | AT piperhansmichael pathophysiologyofsevereischemicmyocardialinjury |