Oxyradicals in medical biology:
Gespeichert in:
| Format: | Elektronisch E-Book |
|---|---|
| Sprache: | English |
| Veröffentlicht: |
Greenwich, Conn.
JAI Press
1998
|
| Schriftenreihe: | Advances in molecular and cell biology
v. 25 |
| Schlagworte: | |
| Online-Zugang: | FAW01 URL des Erstveröffentlichers |
| Beschreibung: | Paralleltitel: Oxyradicals in molecular biology Includes bibliographical references and index The rapid expansion of the area of free radical biology in the last 25 years has occurred within a framework of assumptions and preconceived notions that has at times directed the course of this movement. The most dominant of these notions has been the view that free radical production is without exception a bad thing, and that the more efficient our elimination of these toxic substances, the better off we will be. The very important observation by Bernard Babior and colleagues in 1973 that activated phagocytes produce superoxide in order to kill micro organisms, served to illustrate that constructive roles are possible for free radicals. For many in the field, however, this merely underscored the deadly nature of oxygen-derived radicals, both from the microbe's point of view and from the host's as well. (Phagocyte-produced superoxide is responsible in part for the tissue injury manifested as inflammation. See Harris and Granger, Chapter 5, and Leff, Hybertson and Repine, Chapter 6.) Mother Nature, however, has a penchant for being able to make a silk purse from a sow's ear. If one is dealt a bad hand, one must simply make the best of it. After two decades of focusing on the destructive side of free radicals, the last few years have begun to reveal a new and finer perspective on free radical metabolism - a role in regulation of cellular function (see Schulze-Osthoff and Baeuerle, Chapter 2). Evidence from a number of sources suggests that an increase in the oxidative status of cell encourages that cell to grow and divide. Increasing the expression of mangnese superoxide dismutase can suppress the malignant phenotype of melanon cells (see Oberley and Oberley, Chapter 3). Oxidative stress beyond a certain poitosis (from the Greek, literally "to fall apart"). Is this suicide response an evolutionary fail-safe device to curtail tumorogenesis? Does oxidative stress-induced apoptosis account for the loss of immune cells in AIDS (see Flores and McCor Chapter 4)? This volume attempts to present the spectrum of roles, both good and bad played by active oxygen species as understood at this point in the evolution of this field of free radical biology |
| Beschreibung: | x, 189 pages |
| ISBN: | 9780080877105 0080877109 0762303794 9780762303793 1281716715 9781281716712 |
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| 500 | |a The rapid expansion of the area of free radical biology in the last 25 years has occurred within a framework of assumptions and preconceived notions that has at times directed the course of this movement. The most dominant of these notions has been the view that free radical production is without exception a bad thing, and that the more efficient our elimination of these toxic substances, the better off we will be. The very important observation by Bernard Babior and colleagues in 1973 that activated phagocytes produce superoxide in order to kill micro organisms, served to illustrate that constructive roles are possible for free radicals. For many in the field, however, this merely underscored the deadly nature of oxygen-derived radicals, both from the microbe's point of view and from the host's as well. (Phagocyte-produced superoxide is responsible in part for the tissue injury manifested as inflammation. | ||
| 500 | |a See Harris and Granger, Chapter 5, and Leff, Hybertson and Repine, Chapter 6.) Mother Nature, however, has a penchant for being able to make a silk purse from a sow's ear. If one is dealt a bad hand, one must simply make the best of it. After two decades of focusing on the destructive side of free radicals, the last few years have begun to reveal a new and finer perspective on free radical metabolism - a role in regulation of cellular function (see Schulze-Osthoff and Baeuerle, Chapter 2). Evidence from a number of sources suggests that an increase in the oxidative status of cell encourages that cell to grow and divide. Increasing the expression of mangnese superoxide dismutase can suppress the malignant phenotype of melanon cells (see Oberley and Oberley, Chapter 3). Oxidative stress beyond a certain poitosis (from the Greek, literally "to fall apart"). | ||
| 500 | |a Is this suicide response an evolutionary fail-safe device to curtail tumorogenesis? Does oxidative stress-induced apoptosis account for the loss of immune cells in AIDS (see Flores and McCor Chapter 4)? This volume attempts to present the spectrum of roles, both good and bad played by active oxygen species as understood at this point in the evolution of this field of free radical biology | ||
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| spelling | Oxyradicals in medical biology series editor, E. Edward Bittar ; guest editor, Joe M. McCord Oxyradicals in molecular biology Greenwich, Conn. JAI Press 1998 x, 189 pages txt rdacontent c rdamedia cr rdacarrier Advances in molecular and cell biology v. 25 Paralleltitel: Oxyradicals in molecular biology Includes bibliographical references and index The rapid expansion of the area of free radical biology in the last 25 years has occurred within a framework of assumptions and preconceived notions that has at times directed the course of this movement. The most dominant of these notions has been the view that free radical production is without exception a bad thing, and that the more efficient our elimination of these toxic substances, the better off we will be. The very important observation by Bernard Babior and colleagues in 1973 that activated phagocytes produce superoxide in order to kill micro organisms, served to illustrate that constructive roles are possible for free radicals. For many in the field, however, this merely underscored the deadly nature of oxygen-derived radicals, both from the microbe's point of view and from the host's as well. (Phagocyte-produced superoxide is responsible in part for the tissue injury manifested as inflammation. See Harris and Granger, Chapter 5, and Leff, Hybertson and Repine, Chapter 6.) Mother Nature, however, has a penchant for being able to make a silk purse from a sow's ear. If one is dealt a bad hand, one must simply make the best of it. After two decades of focusing on the destructive side of free radicals, the last few years have begun to reveal a new and finer perspective on free radical metabolism - a role in regulation of cellular function (see Schulze-Osthoff and Baeuerle, Chapter 2). Evidence from a number of sources suggests that an increase in the oxidative status of cell encourages that cell to grow and divide. Increasing the expression of mangnese superoxide dismutase can suppress the malignant phenotype of melanon cells (see Oberley and Oberley, Chapter 3). Oxidative stress beyond a certain poitosis (from the Greek, literally "to fall apart"). Is this suicide response an evolutionary fail-safe device to curtail tumorogenesis? Does oxidative stress-induced apoptosis account for the loss of immune cells in AIDS (see Flores and McCor Chapter 4)? This volume attempts to present the spectrum of roles, both good and bad played by active oxygen species as understood at this point in the evolution of this field of free radical biology Active oxygen / Physiological effect Cytology Free radicals (Chemistry) Molecular biology Oxygen Pathology, Cellular SCIENCE / Life Sciences / Biochemistry bisacsh Active oxygen in the body fast Active oxygen / Physiological effect fast Free radical reactions fast Free radicals (Chemistry) fast Active oxygen Physiological effect Active oxygen in the body Free radical reactions Cytotoxizität (DE-588)4193696-6 gnd rswk-swf Sauerstoffradikal (DE-588)4179211-7 gnd rswk-swf Pathobiochemie (DE-588)4044888-5 gnd rswk-swf 1\p (DE-588)4143413-4 Aufsatzsammlung gnd-content Sauerstoffradikal (DE-588)4179211-7 s Cytotoxizität (DE-588)4193696-6 s 2\p DE-604 Pathobiochemie (DE-588)4044888-5 s 3\p DE-604 McCord, Joe M. Sonstige oth Bittar, E. E. Sonstige oth http://www.sciencedirect.com/science/bookseries/15692558/25 Verlag URL des Erstveröffentlichers Volltext 1\p cgwrk 20201028 DE-101 https://d-nb.info/provenance/plan#cgwrk 2\p cgwrk 20201028 DE-101 https://d-nb.info/provenance/plan#cgwrk 3\p cgwrk 20201028 DE-101 https://d-nb.info/provenance/plan#cgwrk |
| spellingShingle | Oxyradicals in medical biology Active oxygen / Physiological effect Cytology Free radicals (Chemistry) Molecular biology Oxygen Pathology, Cellular SCIENCE / Life Sciences / Biochemistry bisacsh Active oxygen in the body fast Active oxygen / Physiological effect fast Free radical reactions fast Free radicals (Chemistry) fast Active oxygen Physiological effect Active oxygen in the body Free radical reactions Cytotoxizität (DE-588)4193696-6 gnd Sauerstoffradikal (DE-588)4179211-7 gnd Pathobiochemie (DE-588)4044888-5 gnd |
| subject_GND | (DE-588)4193696-6 (DE-588)4179211-7 (DE-588)4044888-5 (DE-588)4143413-4 |
| title | Oxyradicals in medical biology |
| title_alt | Oxyradicals in molecular biology |
| title_auth | Oxyradicals in medical biology |
| title_exact_search | Oxyradicals in medical biology |
| title_full | Oxyradicals in medical biology series editor, E. Edward Bittar ; guest editor, Joe M. McCord |
| title_fullStr | Oxyradicals in medical biology series editor, E. Edward Bittar ; guest editor, Joe M. McCord |
| title_full_unstemmed | Oxyradicals in medical biology series editor, E. Edward Bittar ; guest editor, Joe M. McCord |
| title_short | Oxyradicals in medical biology |
| title_sort | oxyradicals in medical biology |
| topic | Active oxygen / Physiological effect Cytology Free radicals (Chemistry) Molecular biology Oxygen Pathology, Cellular SCIENCE / Life Sciences / Biochemistry bisacsh Active oxygen in the body fast Active oxygen / Physiological effect fast Free radical reactions fast Free radicals (Chemistry) fast Active oxygen Physiological effect Active oxygen in the body Free radical reactions Cytotoxizität (DE-588)4193696-6 gnd Sauerstoffradikal (DE-588)4179211-7 gnd Pathobiochemie (DE-588)4044888-5 gnd |
| topic_facet | Active oxygen / Physiological effect Cytology Free radicals (Chemistry) Molecular biology Oxygen Pathology, Cellular SCIENCE / Life Sciences / Biochemistry Active oxygen in the body Free radical reactions Active oxygen Physiological effect Cytotoxizität Sauerstoffradikal Pathobiochemie Aufsatzsammlung |
| url | http://www.sciencedirect.com/science/bookseries/15692558/25 |
| work_keys_str_mv | AT mccordjoem oxyradicalsinmedicalbiology AT bittaree oxyradicalsinmedicalbiology AT mccordjoem oxyradicalsinmolecularbiology AT bittaree oxyradicalsinmolecularbiology |