Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation:
Gespeichert in:
| Format: | Elektronisch E-Book |
|---|---|
| Sprache: | English |
| Veröffentlicht: |
Greenwich, CT
JAI Press
©1998
|
| Schlagworte: | |
| Online-Zugang: | FAW01 URL des Erstveröffentlichers |
| Beschreibung: | Includes bibliographical references and index Living organisms exhibit specific responses when confronted with sudden changes in their environmental conditions. The ability of the cells to acclimate to their new environment is the integral driving force for adaptive modification of the cells. Such adaptation involves a number of cellular and biochemical alteration including metabolic homeostasis and reprogramming of gene expression. Changes in metabolic pathways are generally short-lived and reversible, while the consequences of gene expression are a long-term process and may lead to permanent alternation in the pattern of adaptive responses. The heart possesses remarkable ability to adapt itself against any stressful situation by increasing resistance to the adverse consequences. Stress composes the foundation of many degenerative heart diseases including atherosclerosis, spasm, thrombosis, cardiomyopathy, and congestive heart failure. Based on the concept that excessive stress may play a crucial role in the pathogenesis of ischemic heart disease, attempts were made to design methods for preventing of myocardial injury. Creation of stress reactions by repeated ischemia and reperfusion or subjecting the hearts to heat or oxidative stress enables them to meet the future stress challenge. Repeated stress exposures adapt the heart to withstand more severe stress reactions probably by upregulating the cellular defense and direct accumulation of intracellular mediators, which presumably constitute the material basis of increased adaptation to stress. Thus, the powerful cardioprotective effect of adaptation is likely to originate at the cellular and molecular levels that compose fundamental processes in the prophylaxis of such diseases. Volume six of the Advances in Organ Biology series contains state-of-the-art reviews on myocardial preservation and cellular adaptation from the leading authorities in this subject |
| Beschreibung: | xi, 275 pages |
| ISBN: | 0762303913 9780762303915 |
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| 245 | 1 | 0 | |a Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation |c guest editor Dipak K. Das |
| 264 | 1 | |a Greenwich, CT |b JAI Press |c ©1998 | |
| 300 | |a xi, 275 pages | ||
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| 500 | |a Includes bibliographical references and index | ||
| 500 | |a Living organisms exhibit specific responses when confronted with sudden changes in their environmental conditions. The ability of the cells to acclimate to their new environment is the integral driving force for adaptive modification of the cells. Such adaptation involves a number of cellular and biochemical alteration including metabolic homeostasis and reprogramming of gene expression. Changes in metabolic pathways are generally short-lived and reversible, while the consequences of gene expression are a long-term process and may lead to permanent alternation in the pattern of adaptive responses. The heart possesses remarkable ability to adapt itself against any stressful situation by increasing resistance to the adverse consequences. Stress composes the foundation of many degenerative heart diseases including atherosclerosis, spasm, thrombosis, cardiomyopathy, and congestive heart failure. Based on the concept that excessive stress may play a crucial role in the pathogenesis of ischemic heart disease, attempts were made to design methods for preventing of myocardial injury. Creation of stress reactions by repeated ischemia and reperfusion or subjecting the hearts to heat or oxidative stress enables them to meet the future stress challenge. Repeated stress exposures adapt the heart to withstand more severe stress reactions probably by upregulating the cellular defense and direct accumulation of intracellular mediators, which presumably constitute the material basis of increased adaptation to stress. Thus, the powerful cardioprotective effect of adaptation is likely to originate at the cellular and molecular levels that compose fundamental processes in the prophylaxis of such diseases. Volume six of the Advances in Organ Biology series contains state-of-the-art reviews on myocardial preservation and cellular adaptation from the leading authorities in this subject | ||
| 650 | 7 | |a Myocardium / Adaptation |2 fast | |
| 650 | 7 | |a Myocardium / Pathophysiology |2 fast | |
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| 650 | 4 | |a Myocardium |x Adaptation | |
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| dewey-full | 612.1/7 |
| dewey-hundreds | 600 - Technology (Applied sciences) |
| dewey-ones | 612 - Human physiology |
| dewey-raw | 612.1/7 |
| dewey-search | 612.1/7 |
| dewey-sort | 3612.1 17 |
| dewey-tens | 610 - Medicine and health |
| discipline | Medizin |
| format | Electronic eBook |
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| id | DE-604.BV044380773 |
| illustrated | Not Illustrated |
| indexdate | 2024-07-10T07:51:20Z |
| institution | BVB |
| isbn | 0762303913 9780762303915 |
| language | English |
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| physical | xi, 275 pages |
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| publishDate | 1998 |
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| publisher | JAI Press |
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| spelling | Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation guest editor Dipak K. Das Greenwich, CT JAI Press ©1998 xi, 275 pages txt rdacontent c rdamedia cr rdacarrier Includes bibliographical references and index Living organisms exhibit specific responses when confronted with sudden changes in their environmental conditions. The ability of the cells to acclimate to their new environment is the integral driving force for adaptive modification of the cells. Such adaptation involves a number of cellular and biochemical alteration including metabolic homeostasis and reprogramming of gene expression. Changes in metabolic pathways are generally short-lived and reversible, while the consequences of gene expression are a long-term process and may lead to permanent alternation in the pattern of adaptive responses. The heart possesses remarkable ability to adapt itself against any stressful situation by increasing resistance to the adverse consequences. Stress composes the foundation of many degenerative heart diseases including atherosclerosis, spasm, thrombosis, cardiomyopathy, and congestive heart failure. Based on the concept that excessive stress may play a crucial role in the pathogenesis of ischemic heart disease, attempts were made to design methods for preventing of myocardial injury. Creation of stress reactions by repeated ischemia and reperfusion or subjecting the hearts to heat or oxidative stress enables them to meet the future stress challenge. Repeated stress exposures adapt the heart to withstand more severe stress reactions probably by upregulating the cellular defense and direct accumulation of intracellular mediators, which presumably constitute the material basis of increased adaptation to stress. Thus, the powerful cardioprotective effect of adaptation is likely to originate at the cellular and molecular levels that compose fundamental processes in the prophylaxis of such diseases. Volume six of the Advances in Organ Biology series contains state-of-the-art reviews on myocardial preservation and cellular adaptation from the leading authorities in this subject Myocardium / Adaptation fast Myocardium / Pathophysiology fast Myocardium Pathophysiology Myocardium Adaptation Das, Dipak Kumar Sonstige oth http://www.sciencedirect.com/science/bookseries/15692590/6 Verlag URL des Erstveröffentlichers Volltext |
| spellingShingle | Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation Myocardium / Adaptation fast Myocardium / Pathophysiology fast Myocardium Pathophysiology Myocardium Adaptation |
| title | Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation |
| title_auth | Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation |
| title_exact_search | Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation |
| title_full | Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation guest editor Dipak K. Das |
| title_fullStr | Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation guest editor Dipak K. Das |
| title_full_unstemmed | Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation guest editor Dipak K. Das |
| title_short | Advances in organ biology, Volume 6, Myocardial preservation and cellular adaptation |
| title_sort | advances in organ biology volume 6 myocardial preservation and cellular adaptation |
| topic | Myocardium / Adaptation fast Myocardium / Pathophysiology fast Myocardium Pathophysiology Myocardium Adaptation |
| topic_facet | Myocardium / Adaptation Myocardium / Pathophysiology Myocardium Pathophysiology Myocardium Adaptation |
| url | http://www.sciencedirect.com/science/bookseries/15692590/6 |
| work_keys_str_mv | AT dasdipakkumar advancesinorganbiologyvolume6myocardialpreservationandcellularadaptation |