Ventricular fibrillation and sudden coronary death:
Gespeichert in:
| 1. Verfasser: | |
|---|---|
| Format: | Buch |
| Sprache: | English |
| Veröffentlicht: |
Boston [u.a.]
Kluwer Acad. Publ.
1999
|
| Schriftenreihe: | Developments in cardiovascular medicine
219 |
| Schlagworte: | |
| Online-Zugang: | Inhaltsverzeichnis |
| Beschreibung: | XXII, 213 S. Ill., graph. Darst. |
| ISBN: | 0792385705 |
Internformat
MARC
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| 100 | 1 | |a Rajskina, Mina |e Verfasser |4 aut | |
| 245 | 1 | 0 | |a Ventricular fibrillation and sudden coronary death |c M. E. Rajskina |
| 264 | 1 | |a Boston [u.a.] |b Kluwer Acad. Publ. |c 1999 | |
| 300 | |a XXII, 213 S. |b Ill., graph. Darst. | ||
| 336 | |b txt |2 rdacontent | ||
| 337 | |b n |2 rdamedia | ||
| 338 | |b nc |2 rdacarrier | ||
| 490 | 1 | |a Developments in cardiovascular medicine |v 219 | |
| 650 | 4 | |a Cardiac arrest | |
| 650 | 4 | |a Coronary Disease |x complications | |
| 650 | 4 | |a Coronary heart disease | |
| 650 | 4 | |a Death, Sudden, Cardiac |x etiology | |
| 650 | 4 | |a Ventricular Fibrillation |x etiology | |
| 650 | 4 | |a Ventricular Fibrillation |x physiopathology | |
| 650 | 4 | |a Ventricular fibrillation | |
| 650 | 0 | 7 | |a Sekundenherztod |0 (DE-588)4180805-8 |2 gnd |9 rswk-swf |
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Datensatz im Suchindex
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|---|---|
| adam_text | Foreword xiii
List of Abbreviations xiv
Introduction xvii
1. The Ventricular Fibrillation and Heart s Blood Supply 1
1.1 The types of cardiac blood supply 1
1.2 Hemodynamic changes 2
1.3 Collateral blood supply of the heart 4
1.4 The size of the ischemic zone 7
1.5 Po2 in the ischemic zone 8
1.6 Conclusion 8
2. Ventricular Fibrillation and Electrophysiological Changes 11
2.1 Role of the VPB in the onset of VF after CAO 11
2.2 Role of ionic current, membrane potentials, automatism, excitability, and
conductivity changes in the appearance of VF after CAO 13
2.2.1 Ischemic changes in the membrane potentials of the heart 15
2.2.2 Ischemic changes of the heart s automatism 16
2.2.3 Ischemic changes of heart s excitability 19
2.2.3.1 The excitation threshold 19
2.2.3.2 The duration of refractory periods 22
2.2.3.3 The latent period 23
2.2.4 Ischemic changes of conductivity 23
2.2.4.1 Speed of excitation propagation in the heart 23
2.2.4.2 Sequence and time dispersion of heart s excitation 24
2.3 Study of mechanisms of ventricular arrhythmia using mathematical modeling
and computer simulation 27
2.3.1 Mathematical model of a cardiac cell 27
2.3.2 Computer simulation study of the initiation of arrhythmia and its
sustaining 30
2.3.3 Spiral waves and action potential characteristics 33
viii VENTRICULAR FIBRILLATION AND SUDDEN DEATH
2.3.4 Modifying action potential parameters to support or termination
spiral waves 35
2.4 Conclusions 35
3. Ventricular Fibrillation and Changes in Metabolism of the Heart after Local Ischemia 37
3.1 Ischemic changes in the ox red equilibrium 39
3.1.1 General oxred potential of myocardium 40
3.1.2 Ox red potential of Lactate/Pyruvate system 40
3.1.3 Oxred potential of the NAD/NAD H system 41
3.1.4 Partial pressure of oxygen in the heart 41
3.1.5 Respiration and oxidative phosphorylation in mitochondria 42
3.1.6 Glycogenolysis changes in CAO 44
3.2 Ischemic disturbances in acid base equilibrium 47
3.2.1 The ischemic changes in extracellular pH 47
3.2.1.1 pH changes in the blood of coronary sinus 47
3.2.1.2 Changes in pH in the coronary vein blood 48
3.2.1.3 Changes in pH of coronary effluate in the time of ischemic
perfusion of an isolated heart s area 49
3.2.1.4 Changes in myocardial pH 49
3.2.2 The ischemic changes in intracellular pH of the myocardium 51
3.3 Ischemic disturbances in ion equilibrium 52
3.3.1 The changes in Na and Cl in the myocardium after CAO 53
3.3.2 The changes of pNa in the coronary sinus blood 53
3.3.3 The changes in content of K+ in blood of coronary vein 53
3.3.4 The changes of K content in coronary effluate 54
3.3.5 Changes in intra and extracellular K+ contents in the myocardium 55
3.3.6 The changes K+ in mitochondria 56
3.3.7 Some mechanisms of ischemic ionic balance disturbances 56
3.3.7.1 The role of ion channels in the loss of K by ischemic cells 56
3.3.7.2 The role of metabolism in the loss of K by ischemic cells 58
3.3.7.3 Role of respiration and oxidative phosphorylation in redistribution
K in the cell 58
3.3.7.4 The role of acid base disturbance in the redistribution K~*~
in the cell 59
3.3.7.5 The role of the glycogenolysis activation in redistribution
K in the cell 60
3.3.7.6 The role of the adrenaline accumulation in an ischemic
heart in K redistribution 60
3.3.7.7 The role of thecompartmentation and decompartmentation
of ions 60
3.4 Conclusion 63
4. Role of the Sympathoadrenal System in the Appearance of Ventricular Fibrillation
after Coronary Artery Occlusion. 65
4.1 Afferent cardiac nerve activity and ventricular fibrillation after coronary artery
occlusion 66
4.2 Afferent activity of the aortal nerve after CAO 71
4.2.1 Changes in systolic discharges 71
4.2.2 Changes in diastolic activity 73
Contents ix
4.2.2.1 The role of the arterial pressure in the increase of diastolic
activity 74
4.2.2.2 The role of metabolic changes in the increase of diastolic
activity 75
4.3 Catecholamine balance in the heart and ventricular fibrillation after coronary
artery occlusion 77
4.3.1 Catecholamine content in the heart in CAO 77
4.3.2 Catecholamine metabolism in the heart after CAO 78
4.3.2.1 Catecholamine synthesis 78
4.3.2.2 Catecholamine catabolism 79
4.3.2.3 Ischemic changes in the catecholamine metabolism in the
heart and their peculiarities in cases with ventricular fibrillation 79
4.3.3 Transport of catecholamines between the heart and blood 80
4.3.3.1 Continuous determination of release of adrenaline and noradrenaline
from the heart via the biological method 81
4.3.3.2 Concentration of adrenaline and noradrenaline in arterial
and venous coronary blood 82
4.3.3.3 Uptake of adrenaline and release of noradrenaline by the
heart 84
4.3.3.4 Possible mechanisms of increases in uptake of A and release
of NA by the heart after coronary artery occlusion 86
4.3.3.5 Measuring the reactivity of the adrenal gland in experiments
with ventricular fibrillation after coronary artery occlusion 90
4.3.3.6 Pharmacological control of uptake of adrenaline by the heart 90
4.4 Conclusions 91
5. Role of Adrenaline in the Mechanisms of Ventricular Fibrillation Onset after Occlusion
of Coronary Artery 95
5.1 Effect of adrenaline on the ox red equilibrium of the heart 95
5.1.1 Effect of adrenaline on the heart s blood supply 96
5.1.2 Effect of adrenaline on the oxygen content, oxred potential and
NAD.H of the myocardium 97
5.1.3 Effect of adrenaline on the respiration and phosphorylation in mitochondria.
98
5.1.4 Influence of adrenaline on glycogenolysis 101
5.2 Effect of adrenaline on the acid base equilibrium. 101
5.2.1 Effect of adrenaline on pH of coronary sinus blood 101
5.2.2 Effect of adrenaline on myocardial pH 102
5.3 Effect of adrenaline on ionic equilibrium 103
5.3.1 Effect of adrenaline on pK, pNa and pCI in the myocardium 104
5.3.2 Effect of adrenaline on K and Na content in the cytoplasma cells 104
5.3.3 Effect of adrenaline on potassium content in the mitochondria. 106
5.3.4 Effect of adrenaline on the K content in the nuclei. 109
5.3.5 Possible mechanisms of adrenaline effect on the ionic exchange in the
myocardium. 109
5.4 Influence of adrenaline on the heart s electrophysiological processes 111
5.5 The comparison of the effects of arrhythmic and non arrhythmic doses of
adrenaline on a metabolism 113
X VENTRICULAR FIBRILLATION AND SUDDEN DEATH
5.6 Comparison of the effects of coronary artery occlusion and adrenaline on
metabolism and electrophysiological processes in the heart. 115
5.6.1 The effect of adrenaline and CAO on oxidation reduction equilibrium 115
5.6.2 The effect of adrenaline and CAO on the glycogenolysis 116
5.6.3 The action of adrenaline and CAO on acid base and ionic equilibrium 117
5.6.4 The effect of adrenaline and CAO on the electrophysiological processes
in the heart 118
6. The Prevention of Ventricular Fibrillation after Coronary Artery Occlusion 121
6.1 The main directions of the antifibrillatory influences 121
6.1.1 Activation of respiration by cytochrome C and hexahydroubiquinone 122
6.1.2 Depression of glycogenolysis by monoiodacetate 122
6.1.3 Control of the acid base equilibrium by buffer TRIS 125
6.1.4 Influences on disturbances in ionic equilibrium by cardioplegic cocktail 126
6.1.4.1 Effects on loss of K+ by myocardial cells 126
6.1.4.2 Effects on retention of Ca2+ by the myocardial cells 129
6.1.4.3 Effects on different mechanisms of antifibrillatory action 129
6.1.5 Influences on cardiac sympathoadrenal control by reserpine, metanephrine
and propranolol 130
6.1.5.1 Exhaustion of noradrenaline stores in the granules by reserpinel31
6.1.5.2 Depression of adrenalin uptake by the heart with metanephrinl31
6.1.5.3 Blockade of /3 adrenoreceptors by propranolol 133
6.1.6 Comparative characteristic of the studied antifibrillatory influences 141
6.2 Method of computer control of the use of antifibrillatory substances 143
6.2.1 Control criterion of AP parameters relaxation 144
6.2.2 The dependence between relaxation coefficient and susceptibility to
circulation of excitation 147
6.2.3 Effect of antiarrhythmic substances on relaxation coefficient 148
6.2.4 Computer control of myocardial action potential based on the values
of relaxation coefficient 149
6.3 The methods of prognostication of ventricular fibrillation 152
6.3.1 The methods of prognostication of ventricular fibrillation by means
of determination of the heart rate variability and baroreflex sensitivity 152
6.3.2 Method of prognostication of ventricular fibrillation by means of
insulin test 153
6.4 Evaluation of effectiveness of antifibrillatory influences 154
6.4.1 Method of postmortem diagnosis of ventricular fibrillation in cases of
sudden death from acute coronary insufficiency in the population 154
6.4.1.1 Postmortem diagnosis of acute coronary insufficiency 155
6.4.1.2 Postmortem diagnosis of ventricular fibrillation 157
6.4.2 Postmortem study of the frequency of sudden death from ventricular
fibrillation out of hospital and in hospital 164
6.5 Conclusions 165
7. Mechanism of Ventricular Fibrillation Onset After Coronary Artery Occlusion 167
7.1 Electrophysiological hypotheses of ventricular fibrillation mechanism 167
7.1.1 Heterotopic automaticity hypotheses 167
7.1.2 Re entry hypothesis 168
7.1.3 Intercellular uncoupling hypothesis 169
Contents xi
7.2 Metabolic hypotheses of ventricular fibrillation 169
7.2.1 Role of the oxygen differential 169
7.2.2 Role of K exit from the cardiac cells 169
7.2.3 Role of fatty acids 170
7.2.4 Role of pH and lactate 170
7.3 Neurohumoral hypotheses of ventricular fibrillation 170
7.3.1 Role of sympathoadrenal system 170
7.3.2 Role of parasympathetic system 170
7.3.3 The role of cAMP 170
7.4 Author s hypothesis based on the systemic approach to study of ventricular
fibrillation mechanisms 171
7.4.1 Sequence and interaction of ischemic changes in the heart preceding
ventricular fibrillation 171
7.4.2 The specifics in metabolic and electrophysiological changes preceding
ventricular fibrillation after coronary artery occlusion 173
7.4.3 The specific changes of heart s sympathoadrenal control, preceding
ventricular fibrillation after coronary artery occlusion 175
7.4.4 Comparison of the effects of coronary artery occlusion and adrenaline
on the metabolic and electrophysiological processes in the heart 177
7.4.5 The prophylaxis of ventricular fibrillation 178
7.5 Ventricular fibrillation as a result of adrenal overcompensation 180
Implications 182
References 183
Subject Index 211
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| indexdate | 2024-07-09T18:35:39Z |
| institution | BVB |
| isbn | 0792385705 |
| language | English |
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| physical | XXII, 213 S. Ill., graph. Darst. |
| publishDate | 1999 |
| publishDateSearch | 1999 |
| publishDateSort | 1999 |
| publisher | Kluwer Acad. Publ. |
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| series | Developments in cardiovascular medicine |
| series2 | Developments in cardiovascular medicine |
| spelling | Rajskina, Mina Verfasser aut Ventricular fibrillation and sudden coronary death M. E. Rajskina Boston [u.a.] Kluwer Acad. Publ. 1999 XXII, 213 S. Ill., graph. Darst. txt rdacontent n rdamedia nc rdacarrier Developments in cardiovascular medicine 219 Cardiac arrest Coronary Disease complications Coronary heart disease Death, Sudden, Cardiac etiology Ventricular Fibrillation etiology Ventricular Fibrillation physiopathology Ventricular fibrillation Sekundenherztod (DE-588)4180805-8 gnd rswk-swf Kammerflimmern (DE-588)4296478-7 gnd rswk-swf Kammerflimmern (DE-588)4296478-7 s Sekundenherztod (DE-588)4180805-8 s DE-604 Developments in cardiovascular medicine 219 (DE-604)BV000000660 219 HBZ Datenaustausch application/pdf http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&local_base=BVB01&doc_number=008778651&sequence=000002&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA Inhaltsverzeichnis |
| spellingShingle | Rajskina, Mina Ventricular fibrillation and sudden coronary death Developments in cardiovascular medicine Cardiac arrest Coronary Disease complications Coronary heart disease Death, Sudden, Cardiac etiology Ventricular Fibrillation etiology Ventricular Fibrillation physiopathology Ventricular fibrillation Sekundenherztod (DE-588)4180805-8 gnd Kammerflimmern (DE-588)4296478-7 gnd |
| subject_GND | (DE-588)4180805-8 (DE-588)4296478-7 |
| title | Ventricular fibrillation and sudden coronary death |
| title_auth | Ventricular fibrillation and sudden coronary death |
| title_exact_search | Ventricular fibrillation and sudden coronary death |
| title_full | Ventricular fibrillation and sudden coronary death M. E. Rajskina |
| title_fullStr | Ventricular fibrillation and sudden coronary death M. E. Rajskina |
| title_full_unstemmed | Ventricular fibrillation and sudden coronary death M. E. Rajskina |
| title_short | Ventricular fibrillation and sudden coronary death |
| title_sort | ventricular fibrillation and sudden coronary death |
| topic | Cardiac arrest Coronary Disease complications Coronary heart disease Death, Sudden, Cardiac etiology Ventricular Fibrillation etiology Ventricular Fibrillation physiopathology Ventricular fibrillation Sekundenherztod (DE-588)4180805-8 gnd Kammerflimmern (DE-588)4296478-7 gnd |
| topic_facet | Cardiac arrest Coronary Disease complications Coronary heart disease Death, Sudden, Cardiac etiology Ventricular Fibrillation etiology Ventricular Fibrillation physiopathology Ventricular fibrillation Sekundenherztod Kammerflimmern |
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