Inflammatory mediators in perinatal health and disease:
Gespeichert in:
| Format: | Buch |
|---|---|
| Sprache: | English |
| Veröffentlicht: |
Philadelphia [u.a.]
Saunders
1995
|
| Schriftenreihe: | Clinics in perinatology
22,2 |
| Schlagworte: | |
| Online-Zugang: | Inhaltsverzeichnis |
| Beschreibung: | XII S., S. 251 - 544 Ill., graph. Darst. |
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Datensatz im Suchindex
| _version_ | 1804124747056807936 |
|---|---|
| adam_text | INFLAMMATORY MEDIATORS IN PERINATAL
HEALTH AND DISEASE
CONTENTS
Preface xiii
Michael S. Caplan and Joseph R. Hageman
An Introduction to the Structure and Function of Inflammatory
Mediators for Clinicians 251
Joseph R. Hageman and Michael S. Caplan
In this article the authors provide a low key introduction to
the classes of mediators discussed in the balance of this issue.
The most important goal is to provide an orientation for clinicians
and to make the transition from the basic science to the clinical
information a smooth one. The emphasis is on the clinical infor¬
mation and on attempts to provide answers as to why this type
of research is relevant to the day to day care of patients.
Platelet Activating Factor: Role in Fetal Lung Development
and Relationship to Normal and Premature Labor 263
Kiwamu Toyoshima, Hisashi Narahara,
Masayuki Furukawa, Rene A. Frenkel, and John M. Johnston
Platelet activating factor (PAF) has been implicated in a number
of physiologic and disease processes. The role of this autacoid in
reproductive biology is now clearly established. The relationship
of PAF to fetal lung maturation and parturition, including some
complications of pregnancy, is stressed in this article. Included in
this discussion are the roles of PAF in premature labor and
pregnancy induced hypertension, with particular emphasis on
necrotizing enterocolitis and the prevention of this disease by
glucocorticoids and breast feeding.
CLINICS IN PERINATOLOGY VOLUME 22 • NUMBER 2 • JUNE 1995 IX
Premature Labor and Intra Amniotic Infection:
Clinical Aspects and Role of the Cytokines in
Diagnosis and Pathophysiology 281
Ricardo Gomez, Fabio Ghezzi, Roberto Romero,
Hernan Mufioz, Jorge E. Tolosa, and Ivan Rojas
The role of infection in the pathogenesis of preterm labor and
delivery, and the specific mechanisms through which this associa¬
tion is demonstrated are presented. Cellular and biochemical
changes that initiate parturition in the setting of infection are
discussed, particularly prostaglandins and cytokines.
Preeclampsia and Related Disorders: Clinical Aspects and
Relevance of Endothelin and Nitric Oxide 343
Steven A. Friedman, Suzanne L. Lubarsky,
Robert A. Ahokas, Alfredo Nova, and Baha M. Sibai
Preeclampsia is a pregnancy specific disorder believed to result
from widespread endothelial dysfunction. Endothelin and nitric
oxide are two potent vasoactive agents of endothelial origin and,
as such, are postulated to play an important role in the pathogen¬
esis of preeclampsia. If these agents are found to be important in
preeclampsia, they will most likely exert their effects locally,
rather than systemically. Future research on the autocrine and
paracrine effects of endothelin and nitric oxide may yield im¬
portant insights into the etiology and pathogenesis of this enig¬
matic disease.
Understanding Prostaglandin Metabolites and
Platelet Activating Factor in the Pathophysiology and
Treatment of the Antiphospholipid Syndrome 357
Richard K. Silver, Alan M. Peaceman, and Diana M. Adams
Prostaglandin metabolites, including thromboxane, prostacyclin,
and platelet activating factor, may participate as intermediaries in
the pathophysiology of the primary antiphospholipid syndrome
(PAPS). In order to determine safe and effective therapies for
patients with PAPS, we must improve our knowledge base and
specific understanding of the mechanisms that link circulating
antiphospholipid antibodies to decidual vasculopathy and fetal
death. A review of current research is presented along with an
evaluation of medical therapy whose rationale for use is consis¬
tent with current evidence.
Causes of Intrauterirte Growth Restriction 375
Mark G. Neerhof
An adequate supply of essential nutrients, the ability of the pla¬
centa to transport the nutrients to the fetus, and the ability of the
fetus to use the nutrients are essential in normal fetal growth.
Factors that compromise fetal growth by their effect on the
X CONTENTS
mother or the fetus result in intrauterine growth restriction
(IUGR). There are many possible causes of IUGR. This article is
a review of the known causes and role that biochemical mediators
play in the pathophysiology of IUGR.
The Role of Nitric Oxide, Endothelin, and Prostaglandins
in the Transition of the Pulmonary Circulation 387
James W. Ziegler, D. Dunbar Ivy, John P. Kinsella, and
Steven H. Abman
The pulmonary endothelial cell regulates fetal and transitional
pulmonary vascular resistance by producing and releasing locally
acting vasodilating and vasoconstricting mediators. Nitric oxide,
endothelin, and the eicosanoids are three endothelial derived
products that play very important roles in effecting change from
the high resistance, low flow pulmonary circulation in utero to
the low resistance, high flow pulmonary circulation postnatally.
This article reviews the roles of these three mediators in the fetal
and transitional circulations. Understanding the mechanisms by
which these mediators influence vascular tone allows for a more
rational approach to disease states associated with abnormal post¬
natal adaptation of the pulmonary circulation.
Persistent Pulmonary Hypertension of the Newborn:
Role of Nitric Oxide and Endothelin in Pathophysiology
and Treatment 405
Robin H. Steinhorn, Susan L. Millard, and
Frederick C. Morin III
Persistent pulmonary hypertension of the newborn (PPHN) re¬
sults in significant morbidity and mortality in otherwise normal
term infants. Safe, effective therapies for PPHN will only be
possible when they can be directed toward the specific defects
producing this condition. In this review, the authors discuss three
different categories of mediators that may play a role in the
normal transition at birth and in the pathophysiology seen in
PPHN: (1) lipid mediators, (2) the peptide endothelin, and (3) the
oxidant radical, nitric oxide. The potential of using the last media¬
tor, nitric oxide, as a treatment for PPHN is under intensive
investigation and is discussed in the final section.
Bronchoalveolar Inflammatory Pathophysiology of
Bronchopulmonary Dysplasia 429
Jerry J. Zimmerman
Because of advances in neonatal intensive care resulting in rescue
of increasingly premature infants, the incidence of bronchopul¬
monary dysplasia (BPD) may actually be increasing. An ex¬
panding database derived from animal and clinical experiments
indicates that pulmonary inflammatory autoinjury involving a
plethora of both cellular and humoral mediators defines the cellu
CONTENTS xi
lar pathophysiology of BPD. Modulation of the bronchoalveolar I
inflammatory response provides a potential novel therapeutic I
arena directly addressing the molecular basis of BPD. j
Patent Ductus Arteriosus: Clinical Relevance of
Prostaglandins and Prostaglandin Inhibitors in PDA
Pathophysiology and Treatment 457
Cathy Hammerman
This article presents a comprehensive review of the patent ductus
arteriosus which so often affects the premature neonate. Included
are a discussion of the unique anatomy and physiology of the
ductus, a review of the mechanisms involved in normal closure,
and factors leading to persistent patency. Various therapeutic
options also are discussed, including some new and innovative
approaches.
Hypoxic Ischemic Encephalopathy: Therapeutic Approaches
Against Microvascular Injury, and Role of Neutrophils,
PAF, and Free Radicals 481
Charles Palmer
The asphyxiated newborn undergoes progressive brain damage
even after resuscitation. This article reviews the microvascular
events that contribute to evolving brain injury. The role of neutro¬
phils, platelet activating factor (PAF), and free radicals is stressed
as pivotal mechanisms of injury and opportunities for therapeutic
intervention.
Inflammatory Mediators and Neonatal Sepsis: Rarely Has So
Little Been Known By So Many About So Much 519
William Meadow and Brian Rudinsky
Many questions remain unanswered (and probably many more
remain unasked) about neonatal sepsis. In addition, many media¬
tors of inflammation have been implicated in neonatal sepsis at
one time or another. The goal of this article is to put together
what the authors view as some of the more important outstand¬
ing questions with what is known about some of the more im¬
portant mediators. We address four questions specifically: (1)
Why is neonatal sepsis different from all other (adult) sepsis? (2)
Is control of pulmonary hypertension the white whale or red
herring in neonatal septic shock? (3) Is sepsis more like dominoes
or like 52 pick up? and (4) Is disruption of Qo2/Vo, coupling a
legitimate clinical concern or a laboratory anomaly?
Index 537
Subscription Information Inside back cover
Xii CONTENTS
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| spelling | Inflammatory mediators in perinatal health and disease Michael S. Caplan ..., guest eds. Philadelphia [u.a.] Saunders 1995 XII S., S. 251 - 544 Ill., graph. Darst. txt rdacontent n rdamedia nc rdacarrier Clinics in perinatology 22,2 Endothélines - Physiologie Facteur d'activation plaquettaire - Physiologie Inflammation - physiopathologie Mediatoren gtt Ontstekingen (geneeskunde) gtt Perinatale geneeskunde gtt Inflammation Mediators Perinatology Cytokine (DE-588)4205701-2 gnd rswk-swf Neugeborenenkrankheit (DE-588)4041779-7 gnd rswk-swf Schwangerschaftsstörung (DE-588)4180321-8 gnd rswk-swf Photopathie (DE-588)4194015-5 gnd rswk-swf (DE-588)4143413-4 Aufsatzsammlung gnd-content Neugeborenenkrankheit (DE-588)4041779-7 s Cytokine (DE-588)4205701-2 s DE-604 Schwangerschaftsstörung (DE-588)4180321-8 s Photopathie (DE-588)4194015-5 s Caplan, Michael S. Sonstige oth Clinics in perinatology 22,2 (DE-604)BV000003382 22,2 HBZ Datenaustausch application/pdf http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&local_base=BVB01&doc_number=006872432&sequence=000002&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA Inhaltsverzeichnis |
| spellingShingle | Inflammatory mediators in perinatal health and disease Clinics in perinatology Endothélines - Physiologie Facteur d'activation plaquettaire - Physiologie Inflammation - physiopathologie Mediatoren gtt Ontstekingen (geneeskunde) gtt Perinatale geneeskunde gtt Inflammation Mediators Perinatology Cytokine (DE-588)4205701-2 gnd Neugeborenenkrankheit (DE-588)4041779-7 gnd Schwangerschaftsstörung (DE-588)4180321-8 gnd Photopathie (DE-588)4194015-5 gnd |
| subject_GND | (DE-588)4205701-2 (DE-588)4041779-7 (DE-588)4180321-8 (DE-588)4194015-5 (DE-588)4143413-4 |
| title | Inflammatory mediators in perinatal health and disease |
| title_auth | Inflammatory mediators in perinatal health and disease |
| title_exact_search | Inflammatory mediators in perinatal health and disease |
| title_full | Inflammatory mediators in perinatal health and disease Michael S. Caplan ..., guest eds. |
| title_fullStr | Inflammatory mediators in perinatal health and disease Michael S. Caplan ..., guest eds. |
| title_full_unstemmed | Inflammatory mediators in perinatal health and disease Michael S. Caplan ..., guest eds. |
| title_short | Inflammatory mediators in perinatal health and disease |
| title_sort | inflammatory mediators in perinatal health and disease |
| topic | Endothélines - Physiologie Facteur d'activation plaquettaire - Physiologie Inflammation - physiopathologie Mediatoren gtt Ontstekingen (geneeskunde) gtt Perinatale geneeskunde gtt Inflammation Mediators Perinatology Cytokine (DE-588)4205701-2 gnd Neugeborenenkrankheit (DE-588)4041779-7 gnd Schwangerschaftsstörung (DE-588)4180321-8 gnd Photopathie (DE-588)4194015-5 gnd |
| topic_facet | Endothélines - Physiologie Facteur d'activation plaquettaire - Physiologie Inflammation - physiopathologie Mediatoren Ontstekingen (geneeskunde) Perinatale geneeskunde Inflammation Mediators Perinatology Cytokine Neugeborenenkrankheit Schwangerschaftsstörung Photopathie Aufsatzsammlung |
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