Benign pancreatic disorders:
Gespeichert in:
| Format: | Buch |
|---|---|
| Sprache: | English |
| Veröffentlicht: |
Philadelphia [u.a]
Saunders
2007
|
| Schriftenreihe: | Surgical clinics of North America
87,6 |
| Schlagworte: | |
| Online-Zugang: | Inhaltsverzeichnis |
| Beschreibung: | XVIII S., S. 1309 - 1541 zahlr. Ill., graph. Darst. |
| ISBN: | 1416051279 9781416051275 |
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| 245 | 1 | 0 | |a Benign pancreatic disorders |c guest ed. Stephen W. Behrman |
| 264 | 1 | |a Philadelphia [u.a] |b Saunders |c 2007 | |
| 300 | |a XVIII S., S. 1309 - 1541 |b zahlr. Ill., graph. Darst. | ||
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| adam_text | BENIGN PANCREATIC DISORDERS
CONTENTS
Foreword xiii
Ronald F. Martin
Preface xvii
Stephen W. Behrman
Pathophysiology of Chronic Pancreatitis 1309
Stephen W. Behrman and Eric S. Fowler
Although the most common causes of chronic pancreatitis have not
changed, it has become clear that a host of modifying biochemical,
inflammatory, neural, and genetic deviations allows the disease to
progress. Alterations in biochemical composition allow calcific
stone formation, whereas various toxins, cytokines, and neuro-
peptides contribute to the progression of fibrosis and pain
production. The basic cellular structure contributing to fibrosis of
the pancreas has been elucidated and factors responsible for its
activation delineated. Of most importance is the recent recognition
of a set of genetic mutations that results in several aberrations of
normal pancreatic physiology, which, in conjunction with other
inciting insults or by themselves, allow the disease to begin and
progress.
The Inflammatory Cascade in Acute Pancreatitis: Relevance
to Clinical Disease 1325
Mohammed Elfar, Lillian W. Gaber, Omaima Sabek,
Craig P. Fischer, and A. Osama Gaber
Acute pancreatitis is an inflammatory condition that is initiated by
the intra pancreatic activation of proteases. Pancreatic enzyme
activation triggers a local and systemic inflammatory response that
is associated with recruitment of inflammatory cells into the
pancreas and a widespread up-regulation of inflammatory markers
in distant tissues.
VOLUME 87 • NUMBER 6 • DECEMBER 2007 vii
Radiologic Assessment of Acute and Chronic Pancreatitis 1341
David H. Kim and Perry J. Pickhardt
The capabilities of various imaging modalities, including CT, MRI,
and ultrasound, have markedly increased over recent years. This
has translated into improved detection and improved character¬
ization of various pathologic processes. This article discusses the
current role of imaging in the evaluation of acute and chronic
pancreatitis. CT remains a major focal point in issues related to
acute pancreatitis, whereas MRI (and magnetic resonance chol-
angiopancreatography) plays a larger role in chronic pancreatitis.
Benign Pancreatic Tumors 1359
Sushanth Reddy and Christopher L. Wolfgang
The goal of this article is to describe the different types of benign
pancreatic neoplasms, methods to distinguish between them, and
treatment options. Pancreatic adenocarcinoma is associated with
specific neoplastic lesions that are similar in radiographic
appearance to some benign lesions. The correct differentiation of
these malignant and premalignant lesions from their benign
counterpart is paramount to their proper management.
Endoscopic Management of Acute and Chronic Pancreatitis 1379
Siriboon Attasaranya, Ayman M. Abdel Aziz,
and Glen A. Lehman
Endoscopic therapy has been increasingly recognized as the effective
therapy in selected patients with acute pancreatitis and chronic
pancreatitis (CP). Utility of endotherapy in various conditions
occurring in acute pancreatitis and CP is discussed. Its efficacy,
limitations, and alternatives are addressed. For the best management
of these complex entities, a multidisciplinary approach involving
expertise in all pancreatic specialties is essential to achieve the goal.
Nutrition Support in Pancreatitis 1403
Caitlin S. Curtis and Kenneth A. Kudsk
Nutrition support is especially important in patients who have
pancreatitis, as these patients have high metabolic needs and are
usually unable to ingest sufficient calories from an oral diet
because of pain or intestinal dysfunction. Clinicians must assess
severity of the disease carefully, as initiation and timing of nutrition
support are crucial. Depending on the severity, early nutrition
support may be unnecessary, while late support ultimately may
lead to worse outcomes. Route of nutrition support also plays an
important role in treatment. The clinician has many alternatives
from which to choose, including enteral nutrition given nasogastri-
cally or nasojejunally, or parenteral nutrition given through a
central line. This article explores the role of nutrition support in the
outcome of pancreatitis and provides guidelines to aid the clinician
in caring for patients who have acute and chronic pancreatitis.
viii CONTENTS
Diagnosis and Management of Sphincter of Oddi Dysfunction
and Pancreas Divisum 1417
James A. Madura II and James A. Madura
Sphincter of Oddi dysfunction and pancreas divisum are very
distinct anatomic abnormalities, yet are diagnosed in similar
clinical situations. While both entities are uncommon, they are
most often discovered during the evaluation of postcholecystec-
tomy syndrome, recurrent idiopathic pancreatitis, and biliary or
pancreatic pain when first line studies are normal. Treatment
consists of surgical sphincteroplasty or endoscopic sphincterotomy
for both diagnoses, which result in reliable relief of symptoms for
most sphincter of Oddi dysfunction patients but less predictable
response in pancreas divisum.
Necrotizing Pancreatitis: Diagnosis and Management 1431
John C. Haney and Theodore N. Pappas
Necrotizing pancreatitis is a severe disease characterized by gland
necrosis and a destructive systemic inflammatory response. Early
management involves aggressive resuscitative and supportive
measures. Outcomes are primarily determined by the presence of
late secondary bacterial infection of the necrotic gland. Early
empiric antibiotics and late surgical necrosectomy in the appro¬
priate setting are the keys to managing these sick patients. With
appropriate management, mortality can be minimized and long-
term quality of life may be restored.
Operative and Nonoperative Management of Pancreatic
Pseudocysts 1447
Simon Bergman and W. Scott Melvin
The management of pancreatic pseudocysts has changed greatly
over the last decade. As laparoscopic and endoscopic techniques
continue to evolve, their use in the treatment of pseudocysts has
gained acceptance, whereas the role of percutaneous drainage has
become more limited. The literature on laparoscopic, endoscopic,
and percutaneous management of pancreatic pseudocyst is
reviewed here and, based on these data, a treatment algorithm is
suggested.
Resectional Therapy for Chronic Pancreatitis 1461
Ronald F. Martin and Michael D. Marion
The main indication for operation in the management of chronic
pancreatitis is medically intractable pain. Other indications include
biliary or pancreatic ductal obstruction (with or without pseudo-
cyst formation), mass effect impinging on other organs or
adversely affecting their function, or diagnostic insecurity regard¬
ing the possibility of periampullary or pancreatic neoplasms. This
article describes the current state of affairs for resectional therapy
for chronic inflammatory conditions of the pancreas.
CONTENTS «
The Role of Total Pancreatectomy and Islet Autotransplantation
for Chronic Pancreatitis 1477
Juan J. Blondet, Annelisa M. Carlson, Takashi Kobayashi,
Tun Jie, Melena Bellin, Bernhard J. Hering, Martin L. Freeman,
Greg J. Beilman, and David E.R. Sutherland
Total pancreatectomy and islet autotransplantation are done for
chronic pancreatitis with intractable pain when other treatment
measures have failed, allowing insulin secretory capacity to be
preserved, minimizing or preventing diabetes, while at the same
time removing the root cause of the pain. Since the first case
in 1977, several series have been published. Pain relief is obtained
in most patients, and insulin independence preserved long term in
about a third, with another third having sufficient beta cell function
so that the surgical diabetes is mild. Islet autotransplantation has
been done with partial or total pancreatectomy for benign and
premalignant conditions. Islet autotransplantation should be used
more widely to preserve beta cell mass in major pancreatic resections.
Management of Internal and External Pancreatic Fistulas 1503
Katherine A. Morgan and David B. Adams
A pancreatic fistula is an uncommon and challenging problem for
the general surgeon. Protean in presentation, the underlying
pathophysiology of a pancreatic duct disruption is consistent.
Several basic principles, when followed, simplify management.
These tenets include medical stabilization and nutritional optimi¬
zation, definition of the underlying duct disorder, and, finally,
definitive management with or without surgery. With appropriate
prompt care, patients can achieve good outcomes.
The Management of Pancreatic Trauma in the Modern Era 1515
Anuradha Subramanian, Christopher J. Dente,
and David V. Feliciano
Pancreatic trauma presents challenging diagnostic and therapeutic
dilemmas to trauma surgeons. Injuries to the pancreas have been
associated with reported morbidity rates approaching 45%. If
treatment is delayed, these rates may increase to 60%. The integrity
of the main pancreatic duct is the most important determinant of
outcome after injury to the pancreas. Undiagnosed ductal
disruptions produce secondary infections, fistulas, fluid collections,
and prolonged stays in the intensive care unit and hospital. This
article analyzes the epidemiology, diagnostic approaches, options
for nonoperative and operative management, and outcome after
blunt and penetrating pancreatic trauma.
Index 1533
X CONTENTS
|
| adam_txt |
BENIGN PANCREATIC DISORDERS
CONTENTS
Foreword xiii
Ronald F. Martin
Preface xvii
Stephen W. Behrman
Pathophysiology of Chronic Pancreatitis 1309
Stephen W. Behrman and Eric S. Fowler
Although the most common causes of chronic pancreatitis have not
changed, it has become clear that a host of modifying biochemical,
inflammatory, neural, and genetic deviations allows the disease to
progress. Alterations in biochemical composition allow calcific
stone formation, whereas various toxins, cytokines, and neuro-
peptides contribute to the progression of fibrosis and pain
production. The basic cellular structure contributing to fibrosis of
the pancreas has been elucidated and factors responsible for its
activation delineated. Of most importance is the recent recognition
of a set of genetic mutations that results in several aberrations of
normal pancreatic physiology, which, in conjunction with other
inciting insults or by themselves, allow the disease to begin and
progress.
The Inflammatory Cascade in Acute Pancreatitis: Relevance
to Clinical Disease 1325
Mohammed Elfar, Lillian W. Gaber, Omaima Sabek,
Craig P. Fischer, and A. Osama Gaber
Acute pancreatitis is an inflammatory condition that is initiated by
the intra pancreatic activation of proteases. Pancreatic enzyme
activation triggers a local and systemic inflammatory response that
is associated with recruitment of inflammatory cells into the
pancreas and a widespread up-regulation of inflammatory markers
in distant tissues.
VOLUME 87 • NUMBER 6 • DECEMBER 2007 vii
Radiologic Assessment of Acute and Chronic Pancreatitis 1341
David H. Kim and Perry J. Pickhardt
The capabilities of various imaging modalities, including CT, MRI,
and ultrasound, have markedly increased over recent years. This
has translated into improved detection and improved character¬
ization of various pathologic processes. This article discusses the
current role of imaging in the evaluation of acute and chronic
pancreatitis. CT remains a major focal point in issues related to
acute pancreatitis, whereas MRI (and magnetic resonance chol-
angiopancreatography) plays a larger role in chronic pancreatitis.
Benign Pancreatic Tumors 1359
Sushanth Reddy and Christopher L. Wolfgang
The goal of this article is to describe the different types of benign
pancreatic neoplasms, methods to distinguish between them, and
treatment options. Pancreatic adenocarcinoma is associated with
specific neoplastic lesions that are similar in radiographic
appearance to some benign lesions. The correct differentiation of
these malignant and premalignant lesions from their benign
counterpart is paramount to their proper management.
Endoscopic Management of Acute and Chronic Pancreatitis 1379
Siriboon Attasaranya, Ayman M. Abdel Aziz,
and Glen A. Lehman
Endoscopic therapy has been increasingly recognized as the effective
therapy in selected patients with acute pancreatitis and chronic
pancreatitis (CP). Utility of endotherapy in various conditions
occurring in acute pancreatitis and CP is discussed. Its efficacy,
limitations, and alternatives are addressed. For the best management
of these complex entities, a multidisciplinary approach involving
expertise in all pancreatic specialties is essential to achieve the goal.
Nutrition Support in Pancreatitis 1403
Caitlin S. Curtis and Kenneth A. Kudsk
Nutrition support is especially important in patients who have
pancreatitis, as these patients have high metabolic needs and are
usually unable to ingest sufficient calories from an oral diet
because of pain or intestinal dysfunction. Clinicians must assess
severity of the disease carefully, as initiation and timing of nutrition
support are crucial. Depending on the severity, early nutrition
support may be unnecessary, while late support ultimately may
lead to worse outcomes. Route of nutrition support also plays an
important role in treatment. The clinician has many alternatives
from which to choose, including enteral nutrition given nasogastri-
cally or nasojejunally, or parenteral nutrition given through a
central line. This article explores the role of nutrition support in the
outcome of pancreatitis and provides guidelines to aid the clinician
in caring for patients who have acute and chronic pancreatitis.
viii CONTENTS
Diagnosis and Management of Sphincter of Oddi Dysfunction
and Pancreas Divisum 1417
James A. Madura II and James A. Madura
Sphincter of Oddi dysfunction and pancreas divisum are very
distinct anatomic abnormalities, yet are diagnosed in similar
clinical situations. While both entities are uncommon, they are
most often discovered during the evaluation of postcholecystec-
tomy syndrome, recurrent idiopathic pancreatitis, and biliary or
pancreatic pain when first line studies are normal. Treatment
consists of surgical sphincteroplasty or endoscopic sphincterotomy
for both diagnoses, which result in reliable relief of symptoms for
most sphincter of Oddi dysfunction patients but less predictable
response in pancreas divisum.
Necrotizing Pancreatitis: Diagnosis and Management 1431
John C. Haney and Theodore N. Pappas
Necrotizing pancreatitis is a severe disease characterized by gland
necrosis and a destructive systemic inflammatory response. Early
management involves aggressive resuscitative and supportive
measures. Outcomes are primarily determined by the presence of
late secondary bacterial infection of the necrotic gland. Early
empiric antibiotics and late surgical necrosectomy in the appro¬
priate setting are the keys to managing these sick patients. With
appropriate management, mortality can be minimized and long-
term quality of life may be restored.
Operative and Nonoperative Management of Pancreatic
Pseudocysts 1447
Simon Bergman and W. Scott Melvin
The management of pancreatic pseudocysts has changed greatly
over the last decade. As laparoscopic and endoscopic techniques
continue to evolve, their use in the treatment of pseudocysts has
gained acceptance, whereas the role of percutaneous drainage has
become more limited. The literature on laparoscopic, endoscopic,
and percutaneous management of pancreatic pseudocyst is
reviewed here and, based on these data, a treatment algorithm is
suggested.
Resectional Therapy for Chronic Pancreatitis 1461
Ronald F. Martin and Michael D. Marion
The main indication for operation in the management of chronic
pancreatitis is medically intractable pain. Other indications include
biliary or pancreatic ductal obstruction (with or without pseudo-
cyst formation), mass effect impinging on other organs or
adversely affecting their function, or diagnostic insecurity regard¬
ing the possibility of periampullary or pancreatic neoplasms. This
article describes the current state of affairs for resectional therapy
for chronic inflammatory conditions of the pancreas.
CONTENTS «
The Role of Total Pancreatectomy and Islet Autotransplantation
for Chronic Pancreatitis 1477
Juan J. Blondet, Annelisa M. Carlson, Takashi Kobayashi,
Tun Jie, Melena Bellin, Bernhard J. Hering, Martin L. Freeman,
Greg J. Beilman, and David E.R. Sutherland
Total pancreatectomy and islet autotransplantation are done for
chronic pancreatitis with intractable pain when other treatment
measures have failed, allowing insulin secretory capacity to be
preserved, minimizing or preventing diabetes, while at the same
time removing the root cause of the pain. Since the first case
in 1977, several series have been published. Pain relief is obtained
in most patients, and insulin independence preserved long term in
about a third, with another third having sufficient beta cell function
so that the surgical diabetes is mild. Islet autotransplantation has
been done with partial or total pancreatectomy for benign and
premalignant conditions. Islet autotransplantation should be used
more widely to preserve beta cell mass in major pancreatic resections.
Management of Internal and External Pancreatic Fistulas 1503
Katherine A. Morgan and David B. Adams
A pancreatic fistula is an uncommon and challenging problem for
the general surgeon. Protean in presentation, the underlying
pathophysiology of a pancreatic duct disruption is consistent.
Several basic principles, when followed, simplify management.
These tenets include medical stabilization and nutritional optimi¬
zation, definition of the underlying duct disorder, and, finally,
definitive management with or without surgery. With appropriate
prompt care, patients can achieve good outcomes.
The Management of Pancreatic Trauma in the Modern Era 1515
Anuradha Subramanian, Christopher J. Dente,
and David V. Feliciano
Pancreatic trauma presents challenging diagnostic and therapeutic
dilemmas to trauma surgeons. Injuries to the pancreas have been
associated with reported morbidity rates approaching 45%. If
treatment is delayed, these rates may increase to 60%. The integrity
of the main pancreatic duct is the most important determinant of
outcome after injury to the pancreas. Undiagnosed ductal
disruptions produce secondary infections, fistulas, fluid collections,
and prolonged stays in the intensive care unit and hospital. This
article analyzes the epidemiology, diagnostic approaches, options
for nonoperative and operative management, and outcome after
blunt and penetrating pancreatic trauma.
Index 1533
X CONTENTS |
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| illustrated | Illustrated |
| index_date | 2024-07-02T19:40:00Z |
| indexdate | 2024-07-09T21:10:43Z |
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| isbn | 1416051279 9781416051275 |
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| physical | XVIII S., S. 1309 - 1541 zahlr. Ill., graph. Darst. |
| publishDate | 2007 |
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| series | Surgical clinics of North America |
| series2 | Surgical clinics of North America |
| spelling | Benign pancreatic disorders guest ed. Stephen W. Behrman Philadelphia [u.a] Saunders 2007 XVIII S., S. 1309 - 1541 zahlr. Ill., graph. Darst. txt rdacontent n rdamedia nc rdacarrier Surgical clinics of North America 87,6 Benigne tumoren gtt Pancreas gtt Pancreas Diseases Behrman, Stephen W. Sonstige oth Surgical clinics of North America 87,6 (DE-604)BV000003239 87,6 HBZ Datenaustausch application/pdf http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&local_base=BVB01&doc_number=016290991&sequence=000002&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA Inhaltsverzeichnis |
| spellingShingle | Benign pancreatic disorders Surgical clinics of North America Benigne tumoren gtt Pancreas gtt Pancreas Diseases |
| title | Benign pancreatic disorders |
| title_auth | Benign pancreatic disorders |
| title_exact_search | Benign pancreatic disorders |
| title_exact_search_txtP | Benign pancreatic disorders |
| title_full | Benign pancreatic disorders guest ed. Stephen W. Behrman |
| title_fullStr | Benign pancreatic disorders guest ed. Stephen W. Behrman |
| title_full_unstemmed | Benign pancreatic disorders guest ed. Stephen W. Behrman |
| title_short | Benign pancreatic disorders |
| title_sort | benign pancreatic disorders |
| topic | Benigne tumoren gtt Pancreas gtt Pancreas Diseases |
| topic_facet | Benigne tumoren Pancreas Pancreas Diseases |
| url | http://bvbr.bib-bvb.de:8991/F?func=service&doc_library=BVB01&local_base=BVB01&doc_number=016290991&sequence=000002&line_number=0001&func_code=DB_RECORDS&service_type=MEDIA |
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