Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis:
Lung fibrosis is a dysregulated or exaggerated tissue repair process resulting in the thickening or scarring of lung tissue. It involves the presence of sustained or repeated exposure to a stressor and intricate dynamics between several inflammatory and immune response cells, and the microenvironmen...
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| Main Author: | |
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| Other Authors: | , , , , , |
| Format: | Electronic eBook |
| Language: | English |
| Published: |
Paris
OECD Publishing
2023
|
| Series: | OECD Series on Adverse Outcome Pathways
no.33 |
| Subjects: | |
| Online Access: | DE-862 DE-863 |
| Summary: | Lung fibrosis is a dysregulated or exaggerated tissue repair process resulting in the thickening or scarring of lung tissue. It involves the presence of sustained or repeated exposure to a stressor and intricate dynamics between several inflammatory and immune response cells, and the microenvironment of the alveolar-capillary region consisting of both immune and non-immune cells, and the lung interstitium. This AOP is applicable to a broad group of stressors of diverse properties e.g. metal dusts, pharmacological products, fibres, microorganisms, chemicals, including novel technology-enabled stressors such as nanomaterials. This AOP is referred to as AOP 173 in the Collaborative Adverse Outcome Pathway Wiki (AOP-Wiki). |
| Physical Description: | 1 Online-Ressource (155 Seiten) 21 x 28cm. |
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| 520 | |a Lung fibrosis is a dysregulated or exaggerated tissue repair process resulting in the thickening or scarring of lung tissue. It involves the presence of sustained or repeated exposure to a stressor and intricate dynamics between several inflammatory and immune response cells, and the microenvironment of the alveolar-capillary region consisting of both immune and non-immune cells, and the lung interstitium. This AOP is applicable to a broad group of stressors of diverse properties e.g. metal dusts, pharmacological products, fibres, microorganisms, chemicals, including novel technology-enabled stressors such as nanomaterials. This AOP is referred to as AOP 173 in the Collaborative Adverse Outcome Pathway Wiki (AOP-Wiki). | ||
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| spelling | Halappanavar, Sabina VerfasserIn aut Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis Sabina, Halappanavar ... [et al] Paris OECD Publishing 2023 1 Online-Ressource (155 Seiten) 21 x 28cm. Text txt rdacontent Computermedien c rdamedia Online-Ressource cr rdacarrier OECD Series on Adverse Outcome Pathways no.33 Lung fibrosis is a dysregulated or exaggerated tissue repair process resulting in the thickening or scarring of lung tissue. It involves the presence of sustained or repeated exposure to a stressor and intricate dynamics between several inflammatory and immune response cells, and the microenvironment of the alveolar-capillary region consisting of both immune and non-immune cells, and the lung interstitium. This AOP is applicable to a broad group of stressors of diverse properties e.g. metal dusts, pharmacological products, fibres, microorganisms, chemicals, including novel technology-enabled stressors such as nanomaterials. This AOP is referred to as AOP 173 in the Collaborative Adverse Outcome Pathway Wiki (AOP-Wiki). Environment Sharma, Monita MitwirkendeR ctb Solorio-Rodriguez, Silvia MitwirkendeR ctb Wallin, Hakan MitwirkendeR ctb Vogel, Ulla MitwirkendeR ctb Sullivan, Kristie MitwirkendeR ctb Clippinger, Amy J... MitwirkendeR ctb |
| spellingShingle | Halappanavar, Sabina Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis Environment |
| title | Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis |
| title_auth | Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis |
| title_exact_search | Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis |
| title_full | Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis Sabina, Halappanavar ... [et al] |
| title_fullStr | Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis Sabina, Halappanavar ... [et al] |
| title_full_unstemmed | Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis Sabina, Halappanavar ... [et al] |
| title_short | Substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis |
| title_sort | substance interaction with the pulmonary resident cell membrane components leading to pulmonary fibrosis |
| topic | Environment |
| topic_facet | Environment |
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