Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain /:
Gespeichert in:
Körperschaften: | , |
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Weitere Verfasser: | , , |
Format: | Elektronisch Tagungsbericht E-Book |
Sprache: | English |
Veröffentlicht: |
Washington, D.C. :
National Academy of Sciences,
[1999]
|
Schriftenreihe: | National Academy of Sciences colloquium series.
|
Schlagworte: | |
Online-Zugang: | Volltext |
Beschreibung: | " ... papers, which were presented at the National Academy of Sciences colloquium "The Neurobiology of Pain," held December 11-13, 1998, at the Arnold and Mabel Beckman Center in Irvine, CA"--Page 7627. "July 1999"--Page 7627. |
Beschreibung: | 1 online resource (pages 7627-7755) : illustrations |
Bibliographie: | Includes bibliographical references. |
ISBN: | 0309569338 9780309569330 |
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111 | 2 | |a National Academy of Sciences Colloquium on the Neurobiology of Pain |d (1998 : |c Irvine, Calif.) |0 http://id.loc.gov/authorities/names/no2004004297 | |
245 | 1 | 0 | |a Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / |c [organized by John Liebeskind, Ronald Dubner and Michael Gold]. |
246 | 3 | 0 | |a Neurobiology of pain |
260 | |a Washington, D.C. : |b National Academy of Sciences, |c [1999] | ||
300 | |a 1 online resource (pages 7627-7755) : |b illustrations | ||
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490 | 1 | |a National Academy of Sciences colloquium series | |
500 | |a " ... papers, which were presented at the National Academy of Sciences colloquium "The Neurobiology of Pain," held December 11-13, 1998, at the Arnold and Mabel Beckman Center in Irvine, CA"--Page 7627. | ||
500 | |a "July 1999"--Page 7627. | ||
504 | |a Includes bibliographical references. | ||
588 | 0 | |a Print version record. | |
505 | 0 | |a COLLOQUIUM ON NEUROBIOLOGY OF PAIN -- NATIONAL ACADEMY OF SCIENCES Colloquium Series -- List of Attendees -- Contents -- The neurobiology of pain -- John C. Liebeskind (1935�1997): A tribute -- Sodium channels and pain -- HYPEREXCITABILITY IN DRG CELLS AFTER INJURY -- MULTIPLE SODIUM CHANNELS IN PRIMARY SENSORY NEURONS -- SODIUM CHANNEL GENE EXPRESSION IS ALTERED AFTER INJURY TO DRG NEURONS -- PHYSIOLOGIC CHANGES ACCOMPANY ALTERED SODIUM GENE EXPRESSION AFTER DRG NEURON INJURY -- NEUROTROPHINS MODULATE SODIUM CHANNEL EXPRESSION IN DRG NEURONS | |
505 | 8 | |a SODIUM CHANNEL EXPRESSION IN INFLAMMATORY PAIN MODELSSODIUM CHANNELS AS MOLECULAR TARGETS IN PAIN RESEARCH -- A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of ... -- EVIDENCE FOR A ROLE FOR PN3 IN THE MEDIATION OF ABNORMAL PAIN BEHAVIORS AFTER NERVE AND TISSUE INJURY -- THE PATHOPHYSIOLOGICAL CONTRIBUTION OF NAN/SNS2 IN PERIPHERAL NERVE INJURY? -- CONCLUSIONS -- Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia -- WHY FOCUS ON NA+ CHANNELS? -- WHY TTX-RESISTANT CHANNELS? | |
505 | 8 | |a CONCLUSIONSCalcium regulation of a slow post-spike hyperpolarization in vagal afferent neurons -- RESULTS -- DISCUSSION -- Ion channels gated by heat -- Causalgia, pathological pain, and adrenergic receptors -- Forebrain mechanisms of nociception and pain: Analysis through imaging -- A visceral pain pathway in the dorsal column of the spinal cord -- ASCENDING PATHWAYS THAT MEDIATE VISCERAL NOCICEPTIVE TRANSMISSION -- MORPHOLOGICAL STUDIES OF THE VISCERAL POSTSYNAPTIC DC SYSTEM -- BEHAVIORAL EVIDENCE FOR A DC VISCERAL PAIN PATHWAY | |
505 | 8 | |a FUNCTIONAL MRI STUDIES IN MONKEYSThe spinal biology in humans and animals of pain states generated by persistent small afferent input -- BEHAVIORAL EFFECTS OF CUTANEOUS STIMULI AFTER INJURY -- ROLE OF SPINAL AND PERIPHERAL SYSTEMS IN THE POST-TISSUE INJURY PAIN STATE -- CHARACTERIZATION OF SEVERAL SPINAL COMPONENTS LEADING TO POSTINJURY PAIN STATES -- Spinal Pharmacology of Facilitated Processing -- SYSTEM INTERACTIONS -- HUMAN SPINAL PROCESSING -- Supraspinal contributions to hyperalgesia -- Neurotrophins and hyperalgesia -- CONCLUSIONS | |
505 | 8 | |a Src, a molecular switch governing gain control of synaptic transmission mediated by N-methyl-D-aspartate receptorsCONCLUSIONS -- Pain perception: Is there a role for primary somatosensory cortex? -- Implications of immune-to-brain communication for sickness and pain -- IMMUNE-TO-BRAIN COMMUNICATION IN SICKNESS -- PAIN AS A NATURAL OUTCOME OF IMMUNE-TO-BRAIN COMMUNICATION -- Role of Spinal Cord Glia in Exaggerated Pain -- CONCLUSIONS AND IMPLICATIONS -- Brain-derived neurotrophic factor is an endogenous modulator of nociceptive responses in the spinal cord | |
650 | 0 | |a Neurobiology |v Congresses. | |
650 | 0 | |a Nociceptors |v Congresses. | |
650 | 0 | |a Pain perception |v Congresses. | |
650 | 0 | |a Sodium channels |v Congresses. | |
650 | 0 | |a Ion channels |v Congresses. | |
650 | 6 | |a Neurobiologie |v Congrès. | |
650 | 6 | |a Nocicepteurs |v Congrès. | |
650 | 6 | |a Perception de la douleur |v Congrès. | |
650 | 6 | |a Canaux à sodium |v Congrès. | |
650 | 6 | |a Canaux ioniques |v Congrès. | |
650 | 7 | |a HEALTH & FITNESS |x Pain Management. |2 bisacsh | |
650 | 7 | |a MEDICAL |x Pain Medicine. |2 bisacsh | |
650 | 7 | |a Ion channels |2 fast | |
650 | 7 | |a Neurobiology |2 fast | |
650 | 7 | |a Nociceptors |2 fast | |
650 | 7 | |a Pain perception |2 fast | |
650 | 7 | |a Sodium channels |2 fast | |
655 | 7 | |a Conference papers and proceedings |2 fast | |
700 | 1 | |a Liebeskind, John C. | |
700 | 1 | |a Dubner, Ronald. | |
700 | 1 | |a Gold, Michael Robert. | |
710 | 2 | |a National Academy of Sciences (U.S.) |0 http://id.loc.gov/authorities/names/n79056514 | |
730 | 0 | |a Proceedings of the National Academy of Sciences of the United States of America. |0 http://id.loc.gov/authorities/names/no2003037749 | |
740 | 0 | |a Colloquium on the Neurobiology of Pain. | |
740 | 0 | |a Colloquium on Neurobiology of Pain. | |
776 | 0 | 8 | |i Print version: |a National Academy of Sciences Colloquium on the Neurobiology of Pain (1998 : Irvine, Calif.). |t Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain. |d Washington, D.C. : National Academy of Sciences, [1999] |w (OCoLC)54038127 |
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DE-BY-FWS_katkey | ZDB-4-EBA-ocm57735171 |
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adam_text | |
any_adam_object | |
author2 | Liebeskind, John C. Dubner, Ronald Gold, Michael Robert |
author2_role | |
author2_variant | j c l jc jcl r d rd m r g mr mrg |
author_corporate | National Academy of Sciences Colloquium on the Neurobiology of Pain Irvine, Calif. National Academy of Sciences (U.S.) |
author_corporate_role | |
author_facet | Liebeskind, John C. Dubner, Ronald Gold, Michael Robert National Academy of Sciences Colloquium on the Neurobiology of Pain Irvine, Calif. National Academy of Sciences (U.S.) |
author_sort | National Academy of Sciences Colloquium on the Neurobiology of Pain Irvine, Calif. |
building | Verbundindex |
bvnumber | localFWS |
callnumber-first | R - Medicine |
callnumber-label | RB127 |
callnumber-raw | RB127 .P37 1999eb |
callnumber-search | RB127 .P37 1999eb |
callnumber-sort | RB 3127 P37 41999EB |
callnumber-subject | RB - Pathology |
collection | ZDB-4-EBA |
contents | COLLOQUIUM ON NEUROBIOLOGY OF PAIN -- NATIONAL ACADEMY OF SCIENCES Colloquium Series -- List of Attendees -- Contents -- The neurobiology of pain -- John C. Liebeskind (1935�1997): A tribute -- Sodium channels and pain -- HYPEREXCITABILITY IN DRG CELLS AFTER INJURY -- MULTIPLE SODIUM CHANNELS IN PRIMARY SENSORY NEURONS -- SODIUM CHANNEL GENE EXPRESSION IS ALTERED AFTER INJURY TO DRG NEURONS -- PHYSIOLOGIC CHANGES ACCOMPANY ALTERED SODIUM GENE EXPRESSION AFTER DRG NEURON INJURY -- NEUROTROPHINS MODULATE SODIUM CHANNEL EXPRESSION IN DRG NEURONS SODIUM CHANNEL EXPRESSION IN INFLAMMATORY PAIN MODELSSODIUM CHANNELS AS MOLECULAR TARGETS IN PAIN RESEARCH -- A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of ... -- EVIDENCE FOR A ROLE FOR PN3 IN THE MEDIATION OF ABNORMAL PAIN BEHAVIORS AFTER NERVE AND TISSUE INJURY -- THE PATHOPHYSIOLOGICAL CONTRIBUTION OF NAN/SNS2 IN PERIPHERAL NERVE INJURY? -- CONCLUSIONS -- Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia -- WHY FOCUS ON NA+ CHANNELS? -- WHY TTX-RESISTANT CHANNELS? CONCLUSIONSCalcium regulation of a slow post-spike hyperpolarization in vagal afferent neurons -- RESULTS -- DISCUSSION -- Ion channels gated by heat -- Causalgia, pathological pain, and adrenergic receptors -- Forebrain mechanisms of nociception and pain: Analysis through imaging -- A visceral pain pathway in the dorsal column of the spinal cord -- ASCENDING PATHWAYS THAT MEDIATE VISCERAL NOCICEPTIVE TRANSMISSION -- MORPHOLOGICAL STUDIES OF THE VISCERAL POSTSYNAPTIC DC SYSTEM -- BEHAVIORAL EVIDENCE FOR A DC VISCERAL PAIN PATHWAY FUNCTIONAL MRI STUDIES IN MONKEYSThe spinal biology in humans and animals of pain states generated by persistent small afferent input -- BEHAVIORAL EFFECTS OF CUTANEOUS STIMULI AFTER INJURY -- ROLE OF SPINAL AND PERIPHERAL SYSTEMS IN THE POST-TISSUE INJURY PAIN STATE -- CHARACTERIZATION OF SEVERAL SPINAL COMPONENTS LEADING TO POSTINJURY PAIN STATES -- Spinal Pharmacology of Facilitated Processing -- SYSTEM INTERACTIONS -- HUMAN SPINAL PROCESSING -- Supraspinal contributions to hyperalgesia -- Neurotrophins and hyperalgesia -- CONCLUSIONS Src, a molecular switch governing gain control of synaptic transmission mediated by N-methyl-D-aspartate receptorsCONCLUSIONS -- Pain perception: Is there a role for primary somatosensory cortex? -- Implications of immune-to-brain communication for sickness and pain -- IMMUNE-TO-BRAIN COMMUNICATION IN SICKNESS -- PAIN AS A NATURAL OUTCOME OF IMMUNE-TO-BRAIN COMMUNICATION -- Role of Spinal Cord Glia in Exaggerated Pain -- CONCLUSIONS AND IMPLICATIONS -- Brain-derived neurotrophic factor is an endogenous modulator of nociceptive responses in the spinal cord |
ctrlnum | (OCoLC)57735171 |
dewey-full | 616.0472 |
dewey-hundreds | 600 - Technology (Applied sciences) |
dewey-ones | 616 - Diseases |
dewey-raw | 616.0472 |
dewey-search | 616.0472 |
dewey-sort | 3616.0472 |
dewey-tens | 610 - Medicine and health |
discipline | Medizin |
format | Electronic Conference Proceeding eBook |
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genre | Conference papers and proceedings fast |
genre_facet | Conference papers and proceedings |
id | ZDB-4-EBA-ocm57735171 |
illustrated | Illustrated |
indexdate | 2024-11-27T13:15:41Z |
institution | BVB |
institution_GND | http://id.loc.gov/authorities/names/no2004004297 http://id.loc.gov/authorities/names/n79056514 |
isbn | 0309569338 9780309569330 |
language | English |
oclc_num | 57735171 |
open_access_boolean | |
owner | MAIN DE-863 DE-BY-FWS |
owner_facet | MAIN DE-863 DE-BY-FWS |
physical | 1 online resource (pages 7627-7755) : illustrations |
psigel | ZDB-4-EBA |
publishDate | 1999 |
publishDateSearch | 1999 |
publishDateSort | 1999 |
publisher | National Academy of Sciences, |
record_format | marc |
series | National Academy of Sciences colloquium series. |
series2 | National Academy of Sciences colloquium series |
spelling | National Academy of Sciences Colloquium on the Neurobiology of Pain (1998 : Irvine, Calif.) http://id.loc.gov/authorities/names/no2004004297 Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / [organized by John Liebeskind, Ronald Dubner and Michael Gold]. Neurobiology of pain Washington, D.C. : National Academy of Sciences, [1999] 1 online resource (pages 7627-7755) : illustrations text txt rdacontent computer c rdamedia online resource cr rdacarrier National Academy of Sciences colloquium series " ... papers, which were presented at the National Academy of Sciences colloquium "The Neurobiology of Pain," held December 11-13, 1998, at the Arnold and Mabel Beckman Center in Irvine, CA"--Page 7627. "July 1999"--Page 7627. Includes bibliographical references. Print version record. COLLOQUIUM ON NEUROBIOLOGY OF PAIN -- NATIONAL ACADEMY OF SCIENCES Colloquium Series -- List of Attendees -- Contents -- The neurobiology of pain -- John C. Liebeskind (1935â€?1997): A tribute -- Sodium channels and pain -- HYPEREXCITABILITY IN DRG CELLS AFTER INJURY -- MULTIPLE SODIUM CHANNELS IN PRIMARY SENSORY NEURONS -- SODIUM CHANNEL GENE EXPRESSION IS ALTERED AFTER INJURY TO DRG NEURONS -- PHYSIOLOGIC CHANGES ACCOMPANY ALTERED SODIUM GENE EXPRESSION AFTER DRG NEURON INJURY -- NEUROTROPHINS MODULATE SODIUM CHANNEL EXPRESSION IN DRG NEURONS SODIUM CHANNEL EXPRESSION IN INFLAMMATORY PAIN MODELSSODIUM CHANNELS AS MOLECULAR TARGETS IN PAIN RESEARCH -- A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of ... -- EVIDENCE FOR A ROLE FOR PN3 IN THE MEDIATION OF ABNORMAL PAIN BEHAVIORS AFTER NERVE AND TISSUE INJURY -- THE PATHOPHYSIOLOGICAL CONTRIBUTION OF NAN/SNS2 IN PERIPHERAL NERVE INJURY? -- CONCLUSIONS -- Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia -- WHY FOCUS ON NA+ CHANNELS? -- WHY TTX-RESISTANT CHANNELS? CONCLUSIONSCalcium regulation of a slow post-spike hyperpolarization in vagal afferent neurons -- RESULTS -- DISCUSSION -- Ion channels gated by heat -- Causalgia, pathological pain, and adrenergic receptors -- Forebrain mechanisms of nociception and pain: Analysis through imaging -- A visceral pain pathway in the dorsal column of the spinal cord -- ASCENDING PATHWAYS THAT MEDIATE VISCERAL NOCICEPTIVE TRANSMISSION -- MORPHOLOGICAL STUDIES OF THE VISCERAL POSTSYNAPTIC DC SYSTEM -- BEHAVIORAL EVIDENCE FOR A DC VISCERAL PAIN PATHWAY FUNCTIONAL MRI STUDIES IN MONKEYSThe spinal biology in humans and animals of pain states generated by persistent small afferent input -- BEHAVIORAL EFFECTS OF CUTANEOUS STIMULI AFTER INJURY -- ROLE OF SPINAL AND PERIPHERAL SYSTEMS IN THE POST-TISSUE INJURY PAIN STATE -- CHARACTERIZATION OF SEVERAL SPINAL COMPONENTS LEADING TO POSTINJURY PAIN STATES -- Spinal Pharmacology of Facilitated Processing -- SYSTEM INTERACTIONS -- HUMAN SPINAL PROCESSING -- Supraspinal contributions to hyperalgesia -- Neurotrophins and hyperalgesia -- CONCLUSIONS Src, a molecular switch governing gain control of synaptic transmission mediated by N-methyl-D-aspartate receptorsCONCLUSIONS -- Pain perception: Is there a role for primary somatosensory cortex? -- Implications of immune-to-brain communication for sickness and pain -- IMMUNE-TO-BRAIN COMMUNICATION IN SICKNESS -- PAIN AS A NATURAL OUTCOME OF IMMUNE-TO-BRAIN COMMUNICATION -- Role of Spinal Cord Glia in Exaggerated Pain -- CONCLUSIONS AND IMPLICATIONS -- Brain-derived neurotrophic factor is an endogenous modulator of nociceptive responses in the spinal cord Neurobiology Congresses. Nociceptors Congresses. Pain perception Congresses. Sodium channels Congresses. Ion channels Congresses. Neurobiologie Congrès. Nocicepteurs Congrès. Perception de la douleur Congrès. Canaux à sodium Congrès. Canaux ioniques Congrès. HEALTH & FITNESS Pain Management. bisacsh MEDICAL Pain Medicine. bisacsh Ion channels fast Neurobiology fast Nociceptors fast Pain perception fast Sodium channels fast Conference papers and proceedings fast Liebeskind, John C. Dubner, Ronald. Gold, Michael Robert. National Academy of Sciences (U.S.) http://id.loc.gov/authorities/names/n79056514 Proceedings of the National Academy of Sciences of the United States of America. http://id.loc.gov/authorities/names/no2003037749 Colloquium on the Neurobiology of Pain. Colloquium on Neurobiology of Pain. Print version: National Academy of Sciences Colloquium on the Neurobiology of Pain (1998 : Irvine, Calif.). Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain. Washington, D.C. : National Academy of Sciences, [1999] (OCoLC)54038127 National Academy of Sciences colloquium series. http://id.loc.gov/authorities/names/n99009085 FWS01 ZDB-4-EBA FWS_PDA_EBA https://search.ebscohost.com/login.aspx?direct=true&scope=site&db=nlebk&AN=123553 Volltext |
spellingShingle | Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / National Academy of Sciences colloquium series. COLLOQUIUM ON NEUROBIOLOGY OF PAIN -- NATIONAL ACADEMY OF SCIENCES Colloquium Series -- List of Attendees -- Contents -- The neurobiology of pain -- John C. Liebeskind (1935â€?1997): A tribute -- Sodium channels and pain -- HYPEREXCITABILITY IN DRG CELLS AFTER INJURY -- MULTIPLE SODIUM CHANNELS IN PRIMARY SENSORY NEURONS -- SODIUM CHANNEL GENE EXPRESSION IS ALTERED AFTER INJURY TO DRG NEURONS -- PHYSIOLOGIC CHANGES ACCOMPANY ALTERED SODIUM GENE EXPRESSION AFTER DRG NEURON INJURY -- NEUROTROPHINS MODULATE SODIUM CHANNEL EXPRESSION IN DRG NEURONS SODIUM CHANNEL EXPRESSION IN INFLAMMATORY PAIN MODELSSODIUM CHANNELS AS MOLECULAR TARGETS IN PAIN RESEARCH -- A comparison of the potential role of the tetrodotoxin-insensitive sodium channels, PN3/SNS and NaN/SNS2, in rat models of ... -- EVIDENCE FOR A ROLE FOR PN3 IN THE MEDIATION OF ABNORMAL PAIN BEHAVIORS AFTER NERVE AND TISSUE INJURY -- THE PATHOPHYSIOLOGICAL CONTRIBUTION OF NAN/SNS2 IN PERIPHERAL NERVE INJURY? -- CONCLUSIONS -- Tetrodotoxin-resistant Na+ currents and inflammatory hyperalgesia -- WHY FOCUS ON NA+ CHANNELS? -- WHY TTX-RESISTANT CHANNELS? CONCLUSIONSCalcium regulation of a slow post-spike hyperpolarization in vagal afferent neurons -- RESULTS -- DISCUSSION -- Ion channels gated by heat -- Causalgia, pathological pain, and adrenergic receptors -- Forebrain mechanisms of nociception and pain: Analysis through imaging -- A visceral pain pathway in the dorsal column of the spinal cord -- ASCENDING PATHWAYS THAT MEDIATE VISCERAL NOCICEPTIVE TRANSMISSION -- MORPHOLOGICAL STUDIES OF THE VISCERAL POSTSYNAPTIC DC SYSTEM -- BEHAVIORAL EVIDENCE FOR A DC VISCERAL PAIN PATHWAY FUNCTIONAL MRI STUDIES IN MONKEYSThe spinal biology in humans and animals of pain states generated by persistent small afferent input -- BEHAVIORAL EFFECTS OF CUTANEOUS STIMULI AFTER INJURY -- ROLE OF SPINAL AND PERIPHERAL SYSTEMS IN THE POST-TISSUE INJURY PAIN STATE -- CHARACTERIZATION OF SEVERAL SPINAL COMPONENTS LEADING TO POSTINJURY PAIN STATES -- Spinal Pharmacology of Facilitated Processing -- SYSTEM INTERACTIONS -- HUMAN SPINAL PROCESSING -- Supraspinal contributions to hyperalgesia -- Neurotrophins and hyperalgesia -- CONCLUSIONS Src, a molecular switch governing gain control of synaptic transmission mediated by N-methyl-D-aspartate receptorsCONCLUSIONS -- Pain perception: Is there a role for primary somatosensory cortex? -- Implications of immune-to-brain communication for sickness and pain -- IMMUNE-TO-BRAIN COMMUNICATION IN SICKNESS -- PAIN AS A NATURAL OUTCOME OF IMMUNE-TO-BRAIN COMMUNICATION -- Role of Spinal Cord Glia in Exaggerated Pain -- CONCLUSIONS AND IMPLICATIONS -- Brain-derived neurotrophic factor is an endogenous modulator of nociceptive responses in the spinal cord Neurobiology Congresses. Nociceptors Congresses. Pain perception Congresses. Sodium channels Congresses. Ion channels Congresses. Neurobiologie Congrès. Nocicepteurs Congrès. Perception de la douleur Congrès. Canaux à sodium Congrès. Canaux ioniques Congrès. HEALTH & FITNESS Pain Management. bisacsh MEDICAL Pain Medicine. bisacsh Ion channels fast Neurobiology fast Nociceptors fast Pain perception fast Sodium channels fast |
title | Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / |
title_alt | Neurobiology of pain Proceedings of the National Academy of Sciences of the United States of America. Colloquium on the Neurobiology of Pain. Colloquium on Neurobiology of Pain. |
title_auth | Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / |
title_exact_search | Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / |
title_full | Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / [organized by John Liebeskind, Ronald Dubner and Michael Gold]. |
title_fullStr | Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / [organized by John Liebeskind, Ronald Dubner and Michael Gold]. |
title_full_unstemmed | Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / [organized by John Liebeskind, Ronald Dubner and Michael Gold]. |
title_short | Papers from a National Academy of Sciences Colloquium on the Neurobiology of Pain / |
title_sort | papers from a national academy of sciences colloquium on the neurobiology of pain |
topic | Neurobiology Congresses. Nociceptors Congresses. Pain perception Congresses. Sodium channels Congresses. Ion channels Congresses. Neurobiologie Congrès. Nocicepteurs Congrès. Perception de la douleur Congrès. Canaux à sodium Congrès. Canaux ioniques Congrès. HEALTH & FITNESS Pain Management. bisacsh MEDICAL Pain Medicine. bisacsh Ion channels fast Neurobiology fast Nociceptors fast Pain perception fast Sodium channels fast |
topic_facet | Neurobiology Congresses. Nociceptors Congresses. Pain perception Congresses. Sodium channels Congresses. Ion channels Congresses. Neurobiologie Congrès. Nocicepteurs Congrès. Perception de la douleur Congrès. Canaux à sodium Congrès. Canaux ioniques Congrès. HEALTH & FITNESS Pain Management. MEDICAL Pain Medicine. Ion channels Neurobiology Nociceptors Pain perception Sodium channels Conference papers and proceedings |
url | https://search.ebscohost.com/login.aspx?direct=true&scope=site&db=nlebk&AN=123553 |
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