SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies:
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| Hauptverfasser: | , , , , , , , |
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| Format: | Elektronisch E-Book |
| Sprache: | English |
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Erlangen ; Nürnberg
Friedrich-Alexander-Universität Erlangen-Nürnberg
2023
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| Online-Zugang: | Volltext Volltext Volltext |
| Beschreibung: | 1 Online-Ressource |
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| spelling | Reuter, Nina Verfasser aut SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies Nina Reuter, Xiaohan Chen, Barbara Kropff, Antonia Sophia Peter, William J. Britt, Michael Mach, Klaus Überla, Marco Thomas Erlangen ; Nürnberg Friedrich-Alexander-Universität Erlangen-Nürnberg 2023 1 Online-Ressource txt rdacontent c rdamedia cr rdacarrier Archivierung/Langzeitarchivierung gewährleistet DE-101 pdager Chen, Xiaohan Verfasser aut Kropff, Barbara Verfasser aut Peter, Antonia Sophia Verfasser aut Britt, William J. Verfasser aut Mach, Michael Verfasser aut Überla, Klaus Verfasser aut Thomas, Marco Verfasser aut Sonderdruck aus Viruses Vol. 15 (2023), article number 1500 10.3390/v15071500 https://open.fau.de/handle/openfau/31710 Verlag kostenfrei Volltext https://nbn-resolving.org/urn:nbn:de:101:1-2409101426546.020755960907 Resolving-System kostenfrei Volltext https://d-nb.info/1341524736/34 Langzeitarchivierung Nationalbibliothek kostenfrei Volltext 1\p emakn 0,08413 20240911 DE-101 https://d-nb.info/provenance/plan#emakn 2\p emasg 0,50325 20240911 DE-101 https://d-nb.info/provenance/plan#emasg 3\p npi 20240910 DE-101 https://d-nb.info/provenance/plan#npi |
| spellingShingle | Reuter, Nina Chen, Xiaohan Kropff, Barbara Peter, Antonia Sophia Britt, William J. Mach, Michael Überla, Klaus Thomas, Marco SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies |
| title | SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies |
| title_auth | SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies |
| title_exact_search | SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies |
| title_full | SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies Nina Reuter, Xiaohan Chen, Barbara Kropff, Antonia Sophia Peter, William J. Britt, Michael Mach, Klaus Überla, Marco Thomas |
| title_fullStr | SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies Nina Reuter, Xiaohan Chen, Barbara Kropff, Antonia Sophia Peter, William J. Britt, Michael Mach, Klaus Überla, Marco Thomas |
| title_full_unstemmed | SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies Nina Reuter, Xiaohan Chen, Barbara Kropff, Antonia Sophia Peter, William J. Britt, Michael Mach, Klaus Überla, Marco Thomas |
| title_short | SARS-CoV-2 Spike Protein Is Capable of Inducing Cell–Cell Fusions Independent from Its Receptor ACE2 and This Activity Can Be Impaired by Furin Inhibitors or a Subset of Monoclonal Antibodies |
| title_sort | sars cov 2 spike protein is capable of inducing cell cell fusions independent from its receptor ace2 and this activity can be impaired by furin inhibitors or a subset of monoclonal antibodies |
| url | https://open.fau.de/handle/openfau/31710 https://nbn-resolving.org/urn:nbn:de:101:1-2409101426546.020755960907 https://d-nb.info/1341524736/34 |
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