Cellular and Molecular Alterations in the Failing Human Heart:
The myocardium in heart failure: Cellular and subcellular alterations in the failing human myocardium. H. Just Medizinische Universitatsklinik Freiburg i. Br., Innere Medizin III - Kardiologie, FRG The syndrome of heart failure continues to be a major challenge to clinicians and scientists. Incidenc...
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| Format: | Elektronisch E-Book |
| Sprache: | English |
| Veröffentlicht: |
Heidelberg
Steinkopff
1992
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| Schlagworte: | |
| Online-Zugang: | UBR01 URL des Erstveröffentlichers |
| Zusammenfassung: | The myocardium in heart failure: Cellular and subcellular alterations in the failing human myocardium. H. Just Medizinische Universitatsklinik Freiburg i. Br., Innere Medizin III - Kardiologie, FRG The syndrome of heart failure continues to be a major challenge to clinicians and scientists. Incidence and mortality of the disease are high, the patient is disabled, and is permanently threatened by the high morbidity and mortality. The clinician faces a syndrome of complex pathophysiology. Multiple causes or underlying disorders of the heart have to be differentiated from heart failure itself, which often results in exceedingly difficult diagnoses. Likewise, prognostication meets with difficulties due to problems in separating influences of the underlying disease and the heart failure syndrome itself. In chronic refractory failure annual mortality may exceed 50%. If aortic stenosis or ischemic cardiomyopathy with main stem lesions are present, this percentage may be even higher. The situation becomes particularly threatening to the patient when the reduction in cardiac performance goes along with complex ventricular arrhythmias. Therapy has remained difficult and of limited effectiveness. Major progress was achieved with the introduction of diuretic substances. Of similar importance was the introduction of va so dilating drugs into the treatment of heart failure. The principle of vasodilation has greatly improved our understanding of the disease, and has brought about a major improvement of symptoms, increase of exercise capacity, and reduc tion of mortality. This is especially true for the introduction of the angiotensin converting enzyme inhibitors |
| Beschreibung: | 1 Online-Ressource (XII, 344 p) |
| ISBN: | 9783642724749 |
| DOI: | 10.1007/978-3-642-72474-9 |
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| spelling | Cellular and Molecular Alterations in the Failing Human Heart edited by G. Hasenfuss, Ch. Holubarsch, H. Just, N. R. Alpert Heidelberg Steinkopff 1992 1 Online-Ressource (XII, 344 p) txt rdacontent c rdamedia cr rdacarrier The myocardium in heart failure: Cellular and subcellular alterations in the failing human myocardium. H. Just Medizinische Universitatsklinik Freiburg i. Br., Innere Medizin III - Kardiologie, FRG The syndrome of heart failure continues to be a major challenge to clinicians and scientists. Incidence and mortality of the disease are high, the patient is disabled, and is permanently threatened by the high morbidity and mortality. The clinician faces a syndrome of complex pathophysiology. Multiple causes or underlying disorders of the heart have to be differentiated from heart failure itself, which often results in exceedingly difficult diagnoses. Likewise, prognostication meets with difficulties due to problems in separating influences of the underlying disease and the heart failure syndrome itself. In chronic refractory failure annual mortality may exceed 50%. If aortic stenosis or ischemic cardiomyopathy with main stem lesions are present, this percentage may be even higher. The situation becomes particularly threatening to the patient when the reduction in cardiac performance goes along with complex ventricular arrhythmias. Therapy has remained difficult and of limited effectiveness. Major progress was achieved with the introduction of diuretic substances. Of similar importance was the introduction of va so dilating drugs into the treatment of heart failure. The principle of vasodilation has greatly improved our understanding of the disease, and has brought about a major improvement of symptoms, increase of exercise capacity, and reduc tion of mortality. This is especially true for the introduction of the angiotensin converting enzyme inhibitors Cardiology Human Physiology Human physiology Molekularpathologie (DE-588)4170396-0 gnd rswk-swf Herzinsuffizienz (DE-588)4024655-3 gnd rswk-swf Herzstoffwechsel (DE-588)4159692-4 gnd rswk-swf Herzmuskelzelle (DE-588)4127327-8 gnd rswk-swf Herzmuskel (DE-588)4024675-9 gnd rswk-swf Störung (DE-588)4133232-5 gnd rswk-swf Herzfehler (DE-588)4024646-2 gnd rswk-swf Cytopathologie (DE-588)4122103-5 gnd rswk-swf (DE-588)1071861417 Konferenzschrift 1991 Gargellen gnd-content Herzinsuffizienz (DE-588)4024655-3 s Herzmuskel (DE-588)4024675-9 s Herzstoffwechsel (DE-588)4159692-4 s Störung (DE-588)4133232-5 s DE-604 Molekularpathologie (DE-588)4170396-0 s Herzmuskelzelle (DE-588)4127327-8 s Cytopathologie (DE-588)4122103-5 s Herzfehler (DE-588)4024646-2 s Hasenfuss, G. edt Holubarsch, Ch edt Just, H. edt Alpert, N. R. edt Erscheint auch als Druck-Ausgabe 9783642724763 Erscheint auch als Druck-Ausgabe 9783798509023 Erscheint auch als Druck-Ausgabe 9783642724756 https://doi.org/10.1007/978-3-642-72474-9 Verlag URL des Erstveröffentlichers Volltext |
| spellingShingle | Cellular and Molecular Alterations in the Failing Human Heart Cardiology Human Physiology Human physiology Molekularpathologie (DE-588)4170396-0 gnd Herzinsuffizienz (DE-588)4024655-3 gnd Herzstoffwechsel (DE-588)4159692-4 gnd Herzmuskelzelle (DE-588)4127327-8 gnd Herzmuskel (DE-588)4024675-9 gnd Störung (DE-588)4133232-5 gnd Herzfehler (DE-588)4024646-2 gnd Cytopathologie (DE-588)4122103-5 gnd |
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| title | Cellular and Molecular Alterations in the Failing Human Heart |
| title_auth | Cellular and Molecular Alterations in the Failing Human Heart |
| title_exact_search | Cellular and Molecular Alterations in the Failing Human Heart |
| title_full | Cellular and Molecular Alterations in the Failing Human Heart edited by G. Hasenfuss, Ch. Holubarsch, H. Just, N. R. Alpert |
| title_fullStr | Cellular and Molecular Alterations in the Failing Human Heart edited by G. Hasenfuss, Ch. Holubarsch, H. Just, N. R. Alpert |
| title_full_unstemmed | Cellular and Molecular Alterations in the Failing Human Heart edited by G. Hasenfuss, Ch. Holubarsch, H. Just, N. R. Alpert |
| title_short | Cellular and Molecular Alterations in the Failing Human Heart |
| title_sort | cellular and molecular alterations in the failing human heart |
| topic | Cardiology Human Physiology Human physiology Molekularpathologie (DE-588)4170396-0 gnd Herzinsuffizienz (DE-588)4024655-3 gnd Herzstoffwechsel (DE-588)4159692-4 gnd Herzmuskelzelle (DE-588)4127327-8 gnd Herzmuskel (DE-588)4024675-9 gnd Störung (DE-588)4133232-5 gnd Herzfehler (DE-588)4024646-2 gnd Cytopathologie (DE-588)4122103-5 gnd |
| topic_facet | Cardiology Human Physiology Human physiology Molekularpathologie Herzinsuffizienz Herzstoffwechsel Herzmuskelzelle Herzmuskel Störung Herzfehler Cytopathologie Konferenzschrift 1991 Gargellen |
| url | https://doi.org/10.1007/978-3-642-72474-9 |
| work_keys_str_mv | AT hasenfussg cellularandmolecularalterationsinthefailinghumanheart AT holubarschch cellularandmolecularalterationsinthefailinghumanheart AT justh cellularandmolecularalterationsinthefailinghumanheart AT alpertnr cellularandmolecularalterationsinthefailinghumanheart |